相似文献
1
Combined effects of pesticides.农药的联合效应
Can Med Assoc J. 1969 Jan 25;100(4):173-9.
2
Perinatal toxicity of pesticides.农药的围产期毒性
Can Med Assoc J. 1969 Jan 25;100(4):167-72.
3
Influence of induction of hepatic microsomal enzymes by phenobarbital on toxicity of organic phosphate insecticides.
Proc Soc Exp Biol Med. 1968 Dec;129(3):699-702. doi: 10.3181/00379727-129-33402.
4
The protective effect of aldrin against the toxicity of organophosphate anticholinesterases.艾氏剂对有机磷酸酯类抗胆碱酯酶毒性的保护作用。
J Pharmacol Exp Ther. 1966 Dec;154(3):613-23.
5
Acute toxicity of combinations of pesticides.农药组合的急性毒性
Toxicol Appl Pharmacol. 1967 May;10(3):586-95. doi: 10.1016/0041-008x(67)90097-x.
6
The interaction of pesticides with other factors.农药与其他因素的相互作用。
Residue Rev. 1967;18:21-103. doi: 10.1007/978-1-4615-8422-3_2.
7
[Personal studies in the field of toxicology of pesticides especially of compounds from the anticholinesterase group].
Arh Hig Rada Toksikol. 1968;19(3):397-405.
8
Phenobarbital and organic solvent toxicity.苯巴比妥和有机溶剂毒性。
Am Ind Hyg Assoc J. 1973 Nov;34(11):487-92. doi: 10.1080/0002889738506886.
9
Occupational exposure to aldrin,2,4-D and 2,4,5-T and its relationship to esterases.
IMS Ind Med Surg. 1971 Sep;40(6):23-7.
10
Glucose feeding exacerbates parathion-induced neurotoxicity.葡萄糖喂养会加剧对硫磷诱导的神经毒性。
J Toxicol Environ Health A. 2001 Jun 22;63(4):253-71. doi: 10.1080/15287390151143659.
引用本文的文献
1
Organophosphorus Compounds at 80: Some Old and New Issues.有机磷化合物 80 年：一些老问题和新问题。
Toxicol Sci. 2018 Mar 1;162(1):24-35. doi: 10.1093/toxsci/kfx266.
2
The toxicity of mixtures of specific organophosphate compounds is modulated by paraoxonase 1 status.对氧磷酶 1 状态调节特定有机磷酸化合物混合物的毒性。
Adv Exp Med Biol. 2010;660:47-60. doi: 10.1007/978-1-60761-350-3_6.
3
Paraoxonase 1 (PON1) modulates the toxicity of mixed organophosphorus compounds.对氧磷酶1（PON1）可调节混合有机磷化合物的毒性。
Toxicol Appl Pharmacol. 2009 Apr 15;236(2):142-53. doi: 10.1016/j.taap.2009.02.001. Epub 2009 Feb 9.
4
Toxic interactions among environmental pollutants: corroborating laboratory observations with human experience.环境污染物之间的毒性相互作用：用人类经验佐证实验室观察结果。
Environ Health Perspect. 1994 Nov;102 Suppl 9(Suppl 9):11-7. doi: 10.1289/ehp.94102s911.
5
Inhibition of microsomal drug metabolism by anticholinesterase insecticides.抗胆碱酯酶杀虫剂对微粒体药物代谢的抑制作用。
Bull Environ Contam Toxicol. 1973 Jan;9(1):4-9. doi: 10.1007/BF01856763.
本文引用的文献
1
Cholinesterase inhibition in vitro by OO-diethyl O-p-nitrophenyl thiophosphate (parathion, E 605).对硫磷（E 605）对体外胆碱酯酶的抑制作用
Biochem J. 1951 Sep;49(4):491-4. doi: 10.1042/bj0490491.
2
EFFECTS OF ACUTE AND CHRONIC DDT ADMINISTRATION ON HEPATIC MICROSOMAL DRUG METABOLISM IN THE RAT.急性和慢性给予滴滴涕对大鼠肝脏微粒体药物代谢的影响
Proc Soc Exp Biol Med. 1963 Nov;114:388-92. doi: 10.3181/00379727-114-28686.
3
Stimulatory effects of chlordane on hepatic microsomal drug metabolism in the rat.氯丹对大鼠肝脏微粒体药物代谢的刺激作用。
Toxicol Appl Pharmacol. 1963 May;5:371-86. doi: 10.1016/0041-008x(63)90096-6.
4
Quantitative measurement of inhibition of the enzymatic detoxification of malathion by EPN (ethyl p-nitrophenyl thionobenzenephosphonate).对EPN（对硝基苯基硫代苯膦酸乙酯）抑制马拉硫磷酶解毒作用的定量测定。
Proc Soc Exp Biol Med. 1957 Dec;96(3):813-8. doi: 10.3181/00379727-96-23617.
5
Marked potentiation in mammalian toxicity from simultaneous administration of two anticholinesterase compounds.同时给予两种抗胆碱酯酶化合物会使哺乳动物毒性显著增强。
J Pharmacol Exp Ther. 1957 Sep;121(1):96-106.
6
Studies on the toxicity and pharmacologic actions of the dimethoxy ester of benzotriazine dithiophosphoric acid (DBD, guthion).苯并三嗪二硫代磷酸二甲酯（DBD，谷硫磷）的毒性及药理作用研究
J Pharmacol Exp Ther. 1957 Feb;119(2):208-18.
7
Studies on ali-esterases. V. Substrate specificity of the esterases of some saprophytic Mycobacteria.脂族酯酶研究。V. 某些腐生分枝杆菌酯酶的底物特异性。
Biochem J. 1957 Feb;65(2):223-32. doi: 10.1042/bj0650223.
8
Pharmacological implications of microsomal enzyme induction.微粒体酶诱导的药理学意义。
Pharmacol Rev. 1967 Sep;19(3):317-66.
9
Quantitative measurement of inhibition of aliesterases, acylamidase, and cholinesterase by EPN and Delnav.EPN和地乐酚对羧酸酯酶、酰胺酶和胆碱酯酶抑制作用的定量测定。
Toxicol Appl Pharmacol. 1968 Mar;12(2):273-84. doi: 10.1016/0041-008x(68)90040-9.
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