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Adrenergic nerve degeneration induced by condensation products of adrenaline and acetaldehyde.

作者信息

Azevedo I, Osswald W

出版信息

Naunyn Schmiedebergs Arch Pharmacol. 1977 Nov;300(2):139-44. doi: 10.1007/BF00505044.

DOI:10.1007/BF00505044
PMID:593435
Abstract
摘要

相似文献

1
Adrenergic nerve degeneration induced by condensation products of adrenaline and acetaldehyde.
Naunyn Schmiedebergs Arch Pharmacol. 1977 Nov;300(2):139-44. doi: 10.1007/BF00505044.
2
Chemical sympathectomy due to tetrahydroisoquinolines derived from adrenaline.
Adv Exp Med Biol. 1980;126:131-6. doi: 10.1007/978-1-4684-3632-7_12.
3
Absence of degeneration of adrenergic neurons after prolonged treatment with debrisoquin.使用异喹胍长期治疗后肾上腺素能神经元未发生退变。
J Pharmacol Exp Ther. 1974 Jun;189(3):646-58.
4
Absence of degeneration of adrenergic neurones after prolonged treatment of newborn rats with debrisoquin.
Acta Pharmacol Toxicol (Copenh). 1975 Feb;36(2):132-44. doi: 10.1111/j.1600-0773.1975.tb00779.x.
5
Degeneration and regrowth of adrenergic nerve fibers in the rat peripheral tissues after 6-hydroxydopamine.6-羟基多巴胺处理后大鼠外周组织中肾上腺素能神经纤维的退变与再生
Can J Physiol Pharmacol. 1971 Apr;49(4):345-55. doi: 10.1139/y71-038.
6
The effects of debrisoquin and bretylium on adrenergic nerves as revealed by fluorescence histochemistry.
J Pharmacol Exp Ther. 1970 Jul;174(1):99-110.
7
6-hydroxydopamine, 6-hydroxydopa and degeneration of adrenergic nerves.
Fed Proc. 1971 May-Jun;30(3):877-85.
8
Hydrocortisone-induced increase in the histochemically demonstrable catecholamine content of sympathetic neurons of the newborn rat.氢化可的松引起新生大鼠交感神经元组织化学可显示的儿茶酚胺含量增加。
Brain Res. 1974 Mar 8;67(3):457-66. doi: 10.1016/0006-8993(74)90494-6.
9
[De- and regeneration of adrenergic nerves in the rat iris following administration of toxic false transmitters].
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Degeneration of adrenergic nerves produced by 6-hydroxydopamine.6-羟基多巴胺导致的肾上腺素能神经退变。
Eur J Pharmacol. 1968 Apr;3(1):89-92. doi: 10.1016/0014-2999(68)90056-3.

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Sympathetic denervation caused by long-term noradrenaline infusions; prevention by desipramine and superoxide dismutase.长期输注去甲肾上腺素引起的交感神经去神经支配;地昔帕明和超氧化物歧化酶的预防作用。
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Nerve terminal degeneration in the rat iris observed following chronic topical 2% epinephrine and 0.1% dipivalyl epinephrine: a quantitative comparison of electron microscopic observations and tissue norepinephrine levels.

本文引用的文献

1
Improved technique for the fluorimetric estimation of catecholamines.儿茶酚胺荧光测定的改进技术。
Acta Physiol Scand. 1961 Apr;51:348-55. doi: 10.1111/j.1748-1716.1961.tb02128.x.
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Electron microscopical studies on axonal degeneration in the rat iris following ganglionectomy.大鼠虹膜神经节切除术后轴突退变的电子显微镜研究。
Am J Anat. 1969 Mar;124(3):341-59. doi: 10.1002/aja.1001240305.
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Alkaloids from catecholamines in adrenal tissue: possible role in alcoholism.肾上腺组织中儿茶酚胺衍生的生物碱:在酒精中毒中的潜在作用。
Graefes Arch Clin Exp Ophthalmol. 1992;230(6):575-9. doi: 10.1007/BF00181781.
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Tetrahydroisoquinoline alkaloids in the adrenal medulla after perfusion with "blood concentrations" of ( 14 C)acetaldehyde.用“血药浓度”的(14C)乙醛灌注后肾上腺髓质中的四氢异喹啉生物碱。
Biochem Pharmacol. 1971 Aug;20(8):1757-61. doi: 10.1016/0006-2952(71)90373-x.
5
Biologic concomitants of alcoholism. 1.酒精中毒的生物学伴随因素。1.
N Engl J Med. 1970 Jul 2;283(1):24-32 contd. doi: 10.1056/NEJM197007022830106.
6
Effects of ethanol on central and peripheral noradrenergic neurons.乙醇对中枢和外周去甲肾上腺素能神经元的影响。
J Pharmacol Exp Ther. 1974 May;189(2):380-91.
7
Effects of length of exposure to and concentration of acetaldehyde on the release of catecholamines.
Biochem Pharmacol. 1974 Jan 15;23(2):223-9. doi: 10.1016/0006-2952(74)90413-4.
8
Tetrahydroisoquinoline alkaloids: in vivo metabolites of L-dopa in man.
Nature. 1973 Feb 16;241(5390):439-43. doi: 10.1038/241439a0.
9
Tetrahydroisoquinoline alkaloids: stimulated secretion from the adrenal medulla.四氢异喹啉生物碱:刺激肾上腺髓质分泌。
J Pharmacol Exp Ther. 1973 Jan;184(1):119-28.
10
The pharmacology of 6-hydroxydopamine.6-羟基多巴胺的药理学
Annu Rev Pharmacol. 1973;13:169-80. doi: 10.1146/annurev.pa.13.040173.001125.