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The adenosine triphosphate content of glucose-6-phosphate dehydrogenase-deficient and normal erythrocytes, including studies of a glucose-6-phosphate dehydrogenase-deficient man with "elevated erythrocytic ATP".

作者信息

Brewer G J, Powell R D

出版信息

J Lab Clin Med. 1966 May;67(5):726-41.

PMID:5946032
Abstract
摘要

相似文献

1
The adenosine triphosphate content of glucose-6-phosphate dehydrogenase-deficient and normal erythrocytes, including studies of a glucose-6-phosphate dehydrogenase-deficient man with "elevated erythrocytic ATP".葡萄糖-6-磷酸脱氢酶缺乏型和正常红细胞的三磷酸腺苷含量，包括对一名患有“红细胞ATP升高”的葡萄糖-6-磷酸脱氢酶缺乏症男性的研究。
J Lab Clin Med. 1966 May;67(5):726-41.
2
HEMOLYTIC EFFECT OF PRIMAQUINE. XVII. HEXOKINASE ACTIVITY OF GLUCOSE-6-PHOSPHATE DEHYDROGENASE-DEFICIENT AND NORMAL ERYTHROCYTES.伯氨喹的溶血作用。十七。葡萄糖-6-磷酸脱氢酶缺乏和正常红细胞的己糖激酶活性。
J Lab Clin Med. 1964 Oct;64:601-12.
3
HEREDITARY HEMOLYTIC DISEASE SECONDARY TO GLUCOSE-6-PHOSPHATE DEHYDROGENASE DEFICIENCY: REPORT OF THREE CASES WITH SPECIAL EMPHASIS ON ATP METABOLISM.葡萄糖-6-磷酸脱氢酶缺乏所致的遗传性溶血性疾病：三例报告并特别强调ATP代谢
Blood. 1964 Apr;23:427-44.
4
THE CARBOHYDRATE INTERMEDIATES OF ERYTHROCYTES DEFICIENT IN GLUCOSE-6-PHOSPHATE DEHYDROGENASE.葡萄糖-6-磷酸脱氢酶缺乏的红细胞中的碳水化合物中间体。
Blood. 1965 Jul;26:82-90.
5
HEXOKINASE ACTIVITY IN NORMAL AND GLUCOSE-6-PHOSPHATE DEHYDROGENASE-DEFICIENT ERYTHROCYTES.正常及葡萄糖-6-磷酸脱氢酶缺乏红细胞中的己糖激酶活性
Nature. 1964 Feb 15;201:684-5. doi: 10.1038/201684a0.
6
Understanding the mechanisms for metabolism-linked hemolytic toxicity of primaquine against glucose 6-phosphate dehydrogenase deficient human erythrocytes: evaluation of eryptotic pathway.了解伯氨喹啉对葡萄糖-6-磷酸脱氢酶缺乏的人红细胞代谢相关溶血性毒性的作用机制：细胞凋亡途径的评价。
Toxicology. 2012 Mar 29;294(1):54-60. doi: 10.1016/j.tox.2012.01.015. Epub 2012 Feb 6.
7
[HEMOLYTIC ANEMIA CAUSED BY GLUCOSE-6-PHOSPHATE DEHYDROGENASE DEFICIENCY].
Sem Hop. 1963 Apr 26;39:996-7.
8
A comparative study on the carbohydrate metabolism of glutathione-deficient, glucose-6-phosphate dehydrogenase-deficient and normal human red cells.谷胱甘肽缺乏、葡萄糖-6-磷酸脱氢酶缺乏和正常人红细胞碳水化合物代谢的比较研究。
Biochim Biophys Acta. 1966 May 26;121(1):187-9. doi: 10.1016/0304-4165(66)90370-9.
9
[NORMAL AND PATHOLOGICAL METABOLISM OF RED CELLS].
Maandschr Kindergeneeskd. 1964 Mar;32:116-27.
10
Instability of reduced glutathione and deficiency of glucose-6-phosphate dehydrogenase activity of red cells in normal Indians.正常印度人中还原型谷胱甘肽的不稳定性及红细胞葡萄糖-6-磷酸脱氢酶活性缺乏
Bull Calcutta Sch Trop Med. 1962 Oct;10:162-3.

引用本文的文献

1
Red cell pyruvate kinase deficiency in neonatal jaundice cases in India.印度新生儿黄疸病例中的红细胞丙酮酸激酶缺乏症
Indian J Pediatr. 2006 Nov;73(11):985-8. doi: 10.1007/BF02758302.
2
Hereditary high-adenosine triphosphate syndrome: study of a new variant.
Biochem Genet. 1981 Oct;19(9-10):1017-22. doi: 10.1007/BF00504264.
3
The relationship between red cell 2,3-diphosphoglycerate and levels of hemoglobin in the human.人体中红细胞2,3 - 二磷酸甘油酸与血红蛋白水平之间的关系。
Proc Natl Acad Sci U S A. 1968 Oct;61(2):756-60. doi: 10.1073/pnas.61.2.756.
4
Genetic and population studies of quantitative levels of adenosine triphosphate in human erythrocytes.人类红细胞中三磷酸腺苷定量水平的遗传与群体研究。
Biochem Genet. 1967 Jun;1(1):25-34. doi: 10.1007/BF00487733.
5
The haemolytic effect of various regimens of primaquine with chloroquine in American Negroes with G6PD deficiency and the lack of an effect of various antimalarial suppressive agents on erythrocyte metabolism.伯氨喹与氯喹不同用药方案对美国G6PD缺乏的黑人的溶血作用以及各种抗疟抑制性药物对红细胞代谢无作用。
Bull World Health Organ. 1967;36(2):303-8.
6
[Influence of chloroquine and primaquine on red cell metabolism (author's transl)].氯喹和伯氨喹对红细胞代谢的影响（作者译）
Klin Wochenschr. 1973 Dec 15;51(24):1185-90. doi: 10.1007/BF01468554.
7
[Leukocyte metabolism. IV. Comparative quantitative determination of NAD and ATP of isolated leukocytes and erythrocytes in chronic and acute human leukemias].[白细胞代谢。IV. 慢性和急性人类白血病中分离白细胞和红细胞的NAD和ATP的比较定量测定]
Klin Wochenschr. 1969 Aug 1;47(15):830-4. doi: 10.1007/BF01882312.
8
Identification of a major locus contributing to erythrocyte 2,3-diphosphoglycerate variability in hooded (Long-Evans) rats.在带帽（长 Evans）大鼠中鉴定出一个导致红细胞 2,3-二磷酸甘油酸变异性的主要基因座。
Genetics. 1977 Apr;85(4):669-79. doi: 10.1093/genetics/85.4.669.
9
The mechanism of 5-methyltetrahydrofolate transport by human erythrocytes.人红细胞转运5-甲基四氢叶酸的机制。
J Clin Invest. 1978 May;61(5):1270-5. doi: 10.1172/JCI109043.