Erway L, Hurley L S, Fraser A
Science. 1966 Jun 24;152(3730):1766-8. doi: 10.1126/science.152.3730.1766.
A specific congenital ataxia may be caused by presence of mutant genes and by manganese deficiency during prenatal development in normal mice. Supplementation of the diet of mutant mice with manganese during prenatal development rectifies the aberrant development, resulting in normal behavior. The congential ataxa results from defective development of the the otoliths.
在正常小鼠中，特定的先天性共济失调可能由突变基因的存在以及产前发育期间锰缺乏引起。在产前发育期间给突变小鼠的饮食补充锰可纠正异常发育，从而产生正常行为。先天性共济失调是由耳石发育缺陷导致的。
