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Human red cell acid phosphatase polymorphism. A study on gene frequency and forensic use of the system in cases of disputed paternity.

作者信息

Fuhrmann W, Lichte K H

出版信息

Humangenetik. 1966;3(2):121-6. doi: 10.1007/BF00291293.

DOI:10.1007/BF00291293
PMID:5984973
Abstract
摘要

相似文献

1
Human red cell acid phosphatase polymorphism. A study on gene frequency and forensic use of the system in cases of disputed paternity.
Humangenetik. 1966;3(2):121-6. doi: 10.1007/BF00291293.
2
Quantitative evidence of a silent gene Po of human red cell acid phosphatase in south Polish population.
Forensic Sci. 1977 Sep-Oct;10(2):109-16. doi: 10.1016/0300-9432(77)90103-0.
3
The red cell acid phosphatase polymorphism in Sweden. Gene frequencies and application to disputed paternity.瑞典的红细胞酸性磷酸酶多态性。基因频率及其在亲子关系鉴定中的应用。
Acta Genet Med Gemellol (Roma). 1971 Jan;20(1):77-81. doi: 10.1017/s1120962300011732.
4
[Human red cell acid phosphatase: a new variant (author's transl)].[人类红细胞酸性磷酸酶:一种新的变异体(作者译)]
Z Rechtsmed. 1973 Aug;73(1):17-21. doi: 10.1007/BF02114769.
5
Human red cell acid phosphatase: quantitative evidence of a silent gene PO, and a Danish population study.
Hum Hered. 1976;26(1):43-58. doi: 10.1159/000152782.
6
Polymorphism of the red cell acid phosphatase in the Swiss population.
Humangenetik. 1970;8(4):354-6. doi: 10.1007/BF00280337.
7
[Forensic importance of spectrophotometric tests of the isoenzyme systems red cell acid phosphatase and glutamatic-pyruvic transaminase in settlement of paternity disputes (author's transl)].[红细胞酸性磷酸酶和谷丙转氨酶同工酶系统分光光度法检测在亲子关系纠纷解决中的法医学重要性(作者译)]
Z Rechtsmed. 1974 Jun 18;74(3):177-80. doi: 10.1007/BF00200586.
8
[The phenotype frequency of erythrocyte acid phosphatases and their importance in the paternity process].[红细胞酸性磷酸酶的表型频率及其在亲子鉴定过程中的重要性]
Wien Med Wochenschr. 1968 Apr 13;118(15):328-9.
9
Red cell esterase D in studies of paternity cases in the United Kingdom.英国亲子鉴定案例研究中的红细胞酯酶D
Vox Sang. 1975;28(5):366-70. doi: 10.1111/j.1423-0410.1975.tb02781.x.
10
[Technic, use and value of erythrocyte acid phosphatase in expert testimony].[红细胞酸性磷酸酶在专家鉴定中的技术、应用及价值]
Beitr Gerichtl Med. 1969;25:213-5.

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European ACP1*C allele has recessive deleterious effects on early life viability.欧洲ACP1*C等位基因对早期生命活力具有隐性有害影响。
Hum Biol. 2004 Dec;76(6):817-35. doi: 10.1353/hub.2005.0023.
3
[On the population genetics of acid phosphatase in erythrocytes (E C 3.1.3.2.): phenotype and allele frequency in Southwestern Germany].

本文引用的文献

1
ACID PHOSPHATASES OF HUMAN RED CELLS: PREDICTED PHENOTYPE CONFORMS TO A GENETIC HYPOTHESIS.人类红细胞的酸性磷酸酶:预测的表型符合遗传假说。
Science. 1964 Sep 11;145(3637):1187-8. doi: 10.1126/science.145.3637.1187.
2
GENETICAL STUDIES ON HUMAN RED CELL ACID PHOSPHATASE.人类红细胞酸性磷酸酶的遗传学研究
Am J Hum Genet. 1964 Mar;16(1):141-54.
3
RED CELL ACID PHOSPHATASE VARIANTS: A NEW HUMAN POLYMORPHISM.红细胞酸性磷酸酶变体:一种新的人类多态性
Humangenetik. 1968;5(3):274-7.
4
Gene frequencies of red cell adenosine deaminase, adenylate kinase, phosphoglucomutase, acid phosphatase and serum alpha-1-Antitrypsin (Pi) in a German population.德国人群中红细胞腺苷脱氨酶、腺苷酸激酶、磷酸葡萄糖变位酶、酸性磷酸酶及血清α-1-抗胰蛋白酶(Pi)的基因频率。
Humangenetik. 1970;10(3):235-43. doi: 10.1007/BF00295786.
5
Disagreements between observed and expected data in erythrocyte acid phosphatase polymorphism. Reference laboratories for enzyme polymorphisms.
Z Rechtsmed. 1971;69(3):191-6. doi: 10.1007/BF02078964.
6
On the phenotype distribution of red cell acid phosphatase in Czechoslovakia. The district of Ceské Budĕjovice.关于捷克斯洛伐克红细胞酸性磷酸酶的表型分布。切斯克布杰约维采地区。
Humangenetik. 1971;13(3):247-9. doi: 10.1007/BF00326951.
7
Geographic and ethnic distribution of some red cell enzymes.某些红细胞酶的地理和种族分布。
Humangenetik. 1972;14(3):204-23. doi: 10.1007/BF00278040.
8
[Red cell enzyme polymorphisms in forensic serology].[法医血清学中的红细胞酶多态性]
Z Rechtsmed. 1971;69(2):83-117. doi: 10.1007/BF02093371.
9
[On the utilization of erythrocyte acid phosphatase polymorphism in paternity evaluation].[关于红细胞酸性磷酸酶多态性在亲子鉴定中的应用]
Dtsch Z Gesamte Gerichtl Med. 1968;64(2):127-46.
Nature. 1963 Sep 7;199:969-71. doi: 10.1038/199969a0.
4
[Population genetics of erythrocyte acid phosphatase].
Humangenetik. 1966;2(4):378-80. doi: 10.1007/BF00396455.