Candia O, Rossi G F, Sekino T
Science. 1967 Feb 10;155(3763):720-2. doi: 10.1126/science.155.3763.720.
Contrary to the results of transecting one half of the rostral pons, unilateral partial lesions of the rostral pons, involving either "specific" or "aspecific" structures, do not prevent the appearance of the desynchronized electroencephalographic patterns of deep sleep in the ipsilateral hemi sphere in cats. This effect, however, is obtained by lesions of the medial as well as lateral tegmental structures of the midbrain. These findings seem to indicate that (i) there is not a single pontine structure or group of structures of crucial importance for the EEG desynchronization of deep sleep; the whole rostral pons appears to contrib ute to the EEG-desynchronizing influ ence; (ii) this influence runs rostrally through the midbrain tegmentum, without following any known fiber pathway.
与横断脑桥吻侧一半的结果相反，猫脑桥吻侧的单侧部分损伤，无论是涉及“特异性”还是“非特异性”结构，都不会阻止同侧半球出现深睡眠时的去同步化脑电图模式。然而，这种效应是通过中脑内侧和外侧被盖结构的损伤获得的。这些发现似乎表明：（i）对于深睡眠脑电图去同步化而言，不存在单个至关重要的脑桥结构或结构组；整个脑桥吻侧似乎都对脑电图去同步化影响有贡献；（ii）这种影响通过中脑被盖向吻侧传递，不遵循任何已知的纤维通路。
