• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

Fine structure of myocardial mitochondria in rats after exercise for one-half to two hours.

作者信息

Laguens R P, Gómez-Dumm C L

出版信息

Circ Res. 1967 Sep;21(3):271-9. doi: 10.1161/01.res.21.3.271.

DOI:10.1161/01.res.21.3.271
PMID:6054010
Abstract
摘要

相似文献

1
Fine structure of myocardial mitochondria in rats after exercise for one-half to two hours.
Circ Res. 1967 Sep;21(3):271-9. doi: 10.1161/01.res.21.3.271.
2
[Ultrastructure of the myocardium under various work regimens].[不同工作模式下心肌的超微结构]
Kardiologiia. 1969 Feb;9(2):10-20.
3
[Ultrastructure of the myocardium following physical exercise, fatigue and rest].[体育锻炼、疲劳和休息后心肌的超微结构]
Biull Eksp Biol Med. 1967 Nov;63(11):134-9.
4
Effect of acriflavin on the fine structure of the heart muscle cell mitochondria of normal and exercised rats.吖啶黄素对正常和运动大鼠心肌细胞线粒体精细结构的影响。
J Mol Cell Cardiol. 1972 Jun;4(3):185-93. doi: 10.1016/0022-2828(72)90056-9.
5
Effect of high-protein diet on rat heart mitochondria after exhaustive exercise.高蛋白饮食对力竭运动后大鼠心脏线粒体的影响。
Am J Physiol. 1974 Apr;226(4):996-1000. doi: 10.1152/ajplegacy.1974.226.4.996.
6
Ultrastructural modifications in rat heart: responses to exercise and training.大鼠心脏的超微结构改变:对运动和训练的反应。
Am J Physiol. 1971 Jun;220(6):1935-40. doi: 10.1152/ajplegacy.1971.220.6.1935.
7
Deoxyribonucleic acid synthesis in the heart mitochondria after acute and exhaustive exercise.急性力竭运动后心脏线粒体中的脱氧核糖核酸合成
Experientia. 1968 Feb 15;24(2):163-4. doi: 10.1007/BF02146962.
8
Ultrastructure of rat heart and liver after exhaustive exercise.
Am J Physiol. 1970 Apr;218(4):1150-5. doi: 10.1152/ajplegacy.1970.218.4.1150.
9
Effect of exhausting exercise on rat heart mitochondria.
Am J Physiol. 1973 Aug;225(2):300-5. doi: 10.1152/ajplegacy.1973.225.2.300.
10
Effect of acute and exhaustive exercise upon the fine structure of heart mitochondria.
Experientia. 1966 Apr 15;22(4):244-6. doi: 10.1007/BF01900936.

引用本文的文献

1
Giant mitochondria in cardiomyocytes: cellular architecture in health and disease.巨大线粒体在心肌细胞中的作用:细胞结构与健康和疾病。
Basic Res Cardiol. 2023 Sep 29;118(1):39. doi: 10.1007/s00395-023-01011-3.
2
Electrically stimulated in vitro heart cell mimic of acute exercise reveals novel immediate cellular responses to exercise: Reduced contractility and metabolism, but maintained calcium cycling and increased myofilament calcium sensitivity.电刺激体外心脏细胞模拟急性运动揭示了运动对细胞的即时新反应:收缩性和代谢降低,但钙循环保持,肌球蛋白钙敏感性增加。
Cell Biochem Funct. 2023 Dec;41(8):1147-1161. doi: 10.1002/cbf.3847. Epub 2023 Sep 4.
3
Oxidative Stress, Mitochondrial Function and Adaptation to Exercise: New Perspectives in Nutrition.
氧化应激、线粒体功能与运动适应:营养领域的新视角
Life (Basel). 2021 Nov 22;11(11):1269. doi: 10.3390/life11111269.
4
Ischemia/Reperfusion Injury following Acute Myocardial Infarction: A Critical Issue for Clinicians and Forensic Pathologists.急性心肌梗死后的缺血/再灌注损伤:临床医生和法医病理学家面临的关键问题。
Mediators Inflamm. 2017;2017:7018393. doi: 10.1155/2017/7018393. Epub 2017 Feb 13.
5
Acute exhaustive aerobic exercise training impair cardiomyocyte function and calcium handling in Sprague-Dawley rats.急性力竭性有氧运动训练损害Sprague-Dawley大鼠的心肌细胞功能和钙处理能力。
PLoS One. 2017 Mar 8;12(3):e0173449. doi: 10.1371/journal.pone.0173449. eCollection 2017.
6
Oxidative-Nitrosative Stress and Myocardial Dysfunctions in Sepsis: Evidence from the Literature and Postmortem Observations.脓毒症中的氧化-亚硝化应激与心肌功能障碍:来自文献及尸检观察的证据
Mediators Inflamm. 2016;2016:3423450. doi: 10.1155/2016/3423450. Epub 2016 May 4.
7
Mitochondrial pathways to cardiac recovery: TFAM.心脏恢复的线粒体途径:线粒体转录因子A
Heart Fail Rev. 2016 Sep;21(5):499-517. doi: 10.1007/s10741-016-9561-8.
8
Redox mechanisms of cardiomyocyte mitochondrial protection.心肌细胞线粒体保护的氧化还原机制。
Front Physiol. 2015 Oct 26;6:291. doi: 10.3389/fphys.2015.00291. eCollection 2015.
9
A TAT-frataxin fusion protein increases lifespan and cardiac function in a conditional Friedreich's ataxia mouse model.TAT- frataxin 融合蛋白可延长条件性弗里德里希共济失调症小鼠模型的寿命并改善心脏功能。
Hum Mol Genet. 2012 Mar 15;21(6):1230-47. doi: 10.1093/hmg/ddr554. Epub 2011 Nov 23.
10
Impaired oxidative phosphorylation in overtrained rat myocardium.过度训练的大鼠心肌中氧化磷酸化受损。
Exp Clin Cardiol. 2010 Winter;15(4):e116-27.