视网膜中3H-甘氨酸的刺激释放。
Stimulated release of 3H-glycine from retina.
作者信息
Bauer B
出版信息
Graefes Arch Clin Exp Ophthalmol. 1982;218(1):30-3. doi: 10.1007/BF02134097.
The release of 3H-glycine was studied from rabbit retinas. Kainic acid (10(-5) M), a glutamate agonist, evoked prompt CA2+-dependent increased release of 3H-glycine. However, nuciferine (10(-4) M) could not suppress the effect of kainic acid. The effect of glutamic acid (10(-5) M) was significantly reduced in the presence of nuciferine (10(-4) M). The GABA analogues, muscimol and THIP, had no effect on the release of 3H-glycine. Aspartate and cystic acid were also without effect.
对兔视网膜中3H-甘氨酸的释放进行了研究。谷氨酸激动剂 kainic 酸（10^(-5) M）可迅速引起依赖钙离子的3H-甘氨酸释放增加。然而，荷叶碱（10^(-4) M）不能抑制 kainic 酸的作用。在存在荷叶碱（10^(-4) M）的情况下，谷氨酸（10^(-5) M）的作用显著降低。GABA类似物蝇蕈醇和 THIP 对3H-甘氨酸的释放没有影响。天冬氨酸和胱氨酸也没有作用。
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