高分子量激肽原或其轻链可保护人血浆激肽释放酶不被血浆蛋白酶抑制剂灭活。 - Suppr

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超能文献

High molecular weight kininogen or its light chain protects human plasma kallikrein from inactivation by plasma protease inhibitors.

作者信息

Schapira M, Scott C F, James A, Silver L D, Kueppers F, James H L, Colman R W

出版信息

Biochemistry. 1982 Feb 2;21(3):567-72. doi: 10.1021/bi00532a024.

DOI:10.1021/bi00532a024

PMID:6175341

Abstract

摘要

相似文献

High molecular weight kininogen or its light chain protects human plasma kallikrein from inactivation by plasma protease inhibitors.高分子量激肽原或其轻链可保护人血浆激肽释放酶不被血浆蛋白酶抑制剂灭活。

Biochemistry. 1982 Feb 2;21(3):567-72. doi: 10.1021/bi00532a024.

High molecular weight kininogen potentiates the heparin-accelerated inhibition of plasma kallikrein by antithrombin: role for antithrombin in the regulation of kallikrein.高分子量激肽原增强抗凝血酶对肝素加速的血浆激肽释放酶的抑制作用：抗凝血酶在激肽释放酶调节中的作用。

Biochemistry. 1993 Nov 16;32(45):12136-47. doi: 10.1021/bi00096a026.

Inactivation of factor XIa by plasma protease inhibitors: predominant role of alpha 1-protease inhibitor and protective effect of high molecular weight kininogen.血浆蛋白酶抑制剂对因子XIa的灭活作用：α1-蛋白酶抑制剂的主要作用及高分子量激肽原的保护作用

J Clin Invest. 1982 Apr;69(4):844-52. doi: 10.1172/jci110524.

Protection of human plasma kallikrein from inactivation by C1 inhibitor and other protease inhibitors. The role of high molecular weight kininogen.保护人血浆激肽释放酶不被C1抑制剂和其他蛋白酶抑制剂灭活。高分子量激肽原的作用。

Biochemistry. 1981 May 12;20(10):2738-43. doi: 10.1021/bi00513a006.

Role of high molecular weight kininogen in modulating the inactivation of human plasma kallikrein by plasma protease inhibitors.高分子量激肽原在调节血浆蛋白酶抑制剂对人血浆激肽释放酶失活作用中的作用。

Adv Exp Med Biol. 1983;156:131-41.

High molecular weight kininogen protects human plasma kallikrein and factor XIa against inactivation by plasma protease inhibitors.高分子量激肽原可保护人血浆激肽释放酶和因子XIa不被血浆蛋白酶抑制剂灭活。

Trans Assoc Am Physicians. 1981;94:190-7.

Inactivation of kallikrein in human plasma.人血浆中激肽释放酶的失活

J Clin Invest. 1983 Jan;71(1):149-58. doi: 10.1172/jci110743.

Significance of the regulation of plasma kallikrein by alpha 2M.α2M对血浆激肽释放酶的调节作用的意义。

Ann N Y Acad Sci. 1994 Sep 10;737:347-67. doi: 10.1111/j.1749-6632.1994.tb44323.x.

Study of the effect of high molecular weight kininogen upon the fluid-phase inactivation of kallikrein by C1 inhibitor.高分子量激肽原对C1抑制剂使激肽释放酶液相失活作用的影响研究。

J Biol Chem. 1986 Nov 15;261(32):14965-8.

Contribution of plasma protease inhibitors to the inactivation of kallikrein in plasma.血浆蛋白酶抑制剂对血浆中激肽释放酶失活的作用。

J Clin Invest. 1982 Feb;69(2):462-8. doi: 10.1172/jci110470.

引用本文的文献

The Influence of Plasma Prekallikrein Oligonucleotide Antisense Therapy on Coagulation and Fibrinolysis Assays: A Post-hoc Analysis.血浆前激肽释放酶寡核苷酸反义疗法对凝血和纤溶检测的影响：一项事后分析

Thromb Haemost. 2022 Dec;122(12):2045-2049. doi: 10.1055/a-1926-2367. Epub 2022 Aug 17.

Severe lung lesions caused by Salmonella are prevented by inhibition of the contact system.通过抑制接触系统可预防由沙门氏菌引起的严重肺部病变。

J Exp Med. 2000 Nov 20;192(10):1415-24. doi: 10.1084/jem.192.10.1415.

Binding of high-molecular-mass kininogen to the Apple 1 domain of factor XI is mediated in part by Val64 and Ile77.高分子量激肽原与因子XI的苹果1结构域的结合部分由缬氨酸64和异亮氨酸77介导。

Biochem J. 1994 Dec 15;304 ( Pt 3)(Pt 3):715-21. doi: 10.1042/bj3040715.

Inactivation of factor XII active fragment in normal plasma. Predominant role of C-1-inhibitor.正常血浆中因子 XII 活性片段的失活。C-1 抑制剂的主要作用。

J Clin Invest. 1984 Jun;73(6):1542-9. doi: 10.1172/JCI111360.

Cleavage of human high molecular weight kininogen markedly enhances its coagulant activity. Evidence that this molecule exists as a procofactor.人高分子量激肽原的裂解显著增强其凝血活性。该分子作为前辅因子存在的证据。

J Clin Invest. 1984 Apr;73(4):954-62. doi: 10.1172/JCI111319.

High-molecular weight kininogen. A secreted platelet protein.高分子量激肽原。一种分泌型血小板蛋白。

J Clin Invest. 1983 May;71(5):1477-89. doi: 10.1172/jci110901.

Surface-mediated defense reactions. The plasma contact activation system.表面介导的防御反应。血浆接触激活系统。

J Clin Invest. 1984 May;73(5):1249-53. doi: 10.1172/JCI111326.

Guinea pig macroalbumin. A major inhibitor of activated Hageman factor in plasma with an alpha 2-macroglobulin-like nature.豚鼠巨白蛋白。血浆中活化的哈格曼因子的主要抑制剂，具有α2-巨球蛋白样性质。

Am J Pathol. 1984 Apr;115(1):57-69.

Inactivation of kallikrein in human plasma.人血浆中激肽释放酶的失活

J Clin Invest. 1983 Jan;71(1):149-58. doi: 10.1172/jci110743.

Failure of guinea pig plasma kallikrein to cause neutrophil migration.

Inflammation. 1986 Dec;10(4):333-45. doi: 10.1007/BF00915818.

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