In conclusion, the earlier reports on the phagocytosis-enhancing effect of interferon have now been widely confirmed. Interferon not only activates non-Fc-receptor-mediated phagocytosis such as colloidal carbon or latex beads, but also stimulates Fc-receptor-mediated ingestion of IgG-coated erythrocytes. However, it should be cautioned that all inducers of interferon do not depend on the interferon they stimulate to activate macrophages. The notion that interferon may be a very nonspecific yet crucial factor in the basic defense mechanisms of the body has been further reinforced.
总之，早期关于干扰素增强吞噬作用的报道现已得到广泛证实。干扰素不仅能激活非Fc受体介导的吞噬作用，如对胶体碳或乳胶珠的吞噬，还能刺激Fc受体介导的对IgG包被红细胞的摄取。然而，需要注意的是，并非所有干扰素诱导剂都依赖于它们所刺激产生的干扰素来激活巨噬细胞。干扰素可能是机体基本防御机制中一个非常非特异性但至关重要的因素这一观点得到了进一步强化。
