Suppr超能文献

箭毒蛙毒素诱导轴突坏死,随后再生。

Batrachotoxin induced axonal necrosis followed by regeneration.

作者信息

Moore G R, Robertson D M, Boegman R J

出版信息

Brain Res. 1983 Nov 21;279(1-2):246-9. doi: 10.1016/0006-8993(83)90185-3.

Abstract

Batrachotoxin (BTX), when applied to peripheral nerve in concentrations sufficient to block impulse transmission and axonal transport, causes axonal necrosis. Quantitation of this phenomenon reveals reduction of both myelinated and unmyelinated fibers 7 days post-BTX injection. Evidence for regeneration correlates with the previously reported recovery of postsynaptic events.

摘要

将足以阻断冲动传导和轴突运输的浓度的箭毒蛙毒素(BTX)应用于外周神经时,会导致轴突坏死。对这一现象进行定量分析发现,在注射BTX后7天,有髓纤维和无髓纤维均减少。再生的证据与先前报道的突触后事件的恢复相关。

相似文献

1
Batrachotoxin induced axonal necrosis followed by regeneration.
Brain Res. 1983 Nov 21;279(1-2):246-9. doi: 10.1016/0006-8993(83)90185-3.
2
Batrachotoxin induced axonal necrosis in peripheral nerves.
Brain Res. 1981 Mar 2;207(2):481-5. doi: 10.1016/0006-8993(81)90383-8.
7
Dependence of batrachotoxin block of axoplasmic transport on sodium.
J Neurobiol. 1982 Nov;13(6):537-49. doi: 10.1002/neu.480130608.
10
Batrachotoxin block of fast axoplasmic transport in mammalian nerve fibers.
Science. 1975 Mar 21;187(4181):1087-9. doi: 10.1126/science.46619.

文献AI研究员

20分钟写一篇综述,助力文献阅读效率提升50倍。

立即体验

用中文搜PubMed

大模型驱动的PubMed中文搜索引擎

马上搜索

文档翻译

学术文献翻译模型,支持多种主流文档格式。

立即体验