Kovaleva L I, Paleev N R, Vinogradova T S, Nikiforova T B
Kardiologiia. 1984 Jan;24(1):51-7.
The differentiation between linked parasystole and extrasystole was based on functional tests with exercise and atropin as stimuli. A total of 116 patients (71 with parasystole and 45 with extrasystole) were investigated. Two mechanisms involved in parasystole with fixed links between ectopic complexes were identified: 1) a mechanism of simple quantitative correlation between two pace-makers, 2) a mechanism of the principal pace-maker being discharged with impulses from the parasystolic one. Extrasystole is based on the trigger mechanism of relationship between ectopic excitation and the impulse from the principal pace-maker. Prolonged ECG recording established limits for the maximum interval of links between monotopic extrasystoles and the interectopic interval for paired extrasystole. Extrasystole was shown to be incapable of generating a pace of its own, i.e. a succession of three or more ectopic complexes of similar morphology.
通过以运动和阿托品作为刺激的功能测试，对蝉联性并行心律和期前收缩进行鉴别。共对116例患者（71例并行心律患者和45例期前收缩患者）进行了研究。确定了异位搏动之间存在固定联系的并行心律所涉及的两种机制：1）两个起搏器之间简单的定量相关机制；2）主导起搏器被并行心律起搏器的冲动激发的机制。期前收缩基于异位兴奋与主导起搏器冲动之间关系的触发机制。延长心电图记录确定了单源性期前收缩之间联系的最大间期以及成对期前收缩的异位搏动间期的限度。结果显示，期前收缩无法产生自身节律，即三个或更多形态相似的异位搏动连续出现。
