Chakraborti P, Bhattacharya S
Experientia. 1978 Jan 15;34(1):136-7. doi: 10.1007/BF01921948.
bTSH augmented the fish thyroid peroxidase activity in a dose-response manner. Thyroxine could not modulate the effect of exogenous bTSH, but it decreased the peroxidase activity in a control system when administered alone. The data therefore suggest similar negative feedback control system for TSH-regulation as operative in the case of mammals.
牛促甲状腺激素(bTSH)以剂量反应方式增强鱼类甲状腺过氧化物酶活性。甲状腺素不能调节外源性bTSH的作用,但单独给药时它会降低控制系统中的过氧化物酶活性。因此,这些数据表明,在TSH调节方面存在与哺乳动物类似的负反馈控制系统。