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摄入氯化汞后长期急性肾衰竭期间的血浆汞含量

Plasma mercury during prolonged acute renal failure after mercuric chloride ingestion.

作者信息

Newton J A, House I M, Volans G N, Goodwin F J

出版信息

Hum Toxicol. 1983 Jul;2(3):535-7. doi: 10.1177/096032718300200310.

Abstract

Serial measurements of plasma mercury were made in a patient with severe and prolonged acute renal failure due to poisoning with mercuric chloride. An initial mercury concentration in whole blood of 1200 micrograms/l (6 mumol/l) was recorded, and recovery of renal function coincided with a fall in plasma mercury concentration to below 100 micrograms/l (0.5 mumol/l). The case demonstrates that survival and recovery of renal function is possible despite very high concentrations of mercury in the blood and oliguric renal failure of nearly six weeks' duration.

摘要

对一名因氯化汞中毒导致严重且持续时间较长的急性肾衰竭患者进行了血浆汞的系列测量。记录到全血中汞的初始浓度为1200微克/升(6微摩尔/升),肾功能的恢复与血浆汞浓度降至100微克/升(0.5微摩尔/升)以下同时发生。该病例表明,尽管血液中汞浓度极高且少尿性肾衰竭持续了近六周,但肾功能仍有可能存活并恢复。

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