Host-virus interactions critical for cellular transformation by polyoma virus and SV40.
作者信息
Rassoulzadegan M, Mougneau E, Perbal B, Gaudray P, Birg F, Cuzin F
出版信息
Cold Spring Harb Symp Quant Biol. 1980;44 Pt 1,:333-42. doi: 10.1101/sqb.1980.044.01.038.
PMID:6253148
Abstract
摘要
相似文献
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Host-virus interactions critical for cellular transformation by polyoma virus and SV40.宿主-病毒相互作用对多瘤病毒和SV40诱导细胞转化至关重要。
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Early mutants of polyoma virus (dl8 and dl23) with altered transformation properties: is polyoma virus middle T antigen a transforming gene product?具有改变的转化特性的多瘤病毒早期突变体（dl8和dl23）：多瘤病毒中T抗原是一种转化基因产物吗？
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Monkey cells transformed by SV40 DNA fragments: flat revertants synthesize large and small T antigens.由SV40 DNA片段转化的猴细胞：扁平回复突变体合成大T抗原和小T抗原。
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Viral and cellular control of the SV40-transformed phenotype.SV40转化表型的病毒和细胞调控
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The transforming genes of SV40 and polyoma.猴空泡病毒40和多瘤病毒的转化基因。
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The nucleotide sequence and genome organization of the polyoma early region: extensive nucleotide and amino acid homology with SV40.多瘤病毒早期区域的核苷酸序列和基因组结构：与猴空泡病毒40在核苷酸和氨基酸水平上具有广泛的同源性。
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Transforming genes of DNA tumor viruses.
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DNAs of simian virus 40 and polyoma direct the synthesis of viral tumor antigens and capsid proteins in Xenopus oocytes.猿猴病毒40和多瘤病毒的DNA在非洲爪蟾卵母细胞中指导病毒肿瘤抗原和衣壳蛋白的合成。
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Organization of T antigens in the polyoma virus genome.多瘤病毒基因组中T抗原的组织方式。
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