猿猴病毒40的一种温度敏感、DNA阳性、非转化突变体的特性鉴定 - Suppr

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Characterization of a temperature-sensitive, DNA-positive, nontransforming mutant of simian virus 40.

作者信息

Cosman D J, Tevethia M J

出版信息

Virology. 1981 Jul 30;112(2):605-24. doi: 10.1016/0042-6822(81)90306-8.

DOI:10.1016/0042-6822(81)90306-8

PMID:6266143

Abstract

摘要

相似文献

Characterization of a temperature-sensitive, DNA-positive, nontransforming mutant of simian virus 40.

Virology. 1981 Jul 30;112(2):605-24. doi: 10.1016/0042-6822(81)90306-8.

Mutant carrying deletions in the two simian virus 40 early genes.

J Virol. 1981 Dec;40(3):625-34. doi: 10.1128/JVI.40.3.625-634.1981.

Viral and cellular control of the SV40-transformed phenotype.

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 1,:343-52. doi: 10.1101/sqb.1980.044.01.039.

A simian virus 40 dl884/tsA58 double mutant is temperature sensitive for abortive transformation.

J Virol. 1983 May;46(2):620-5. doi: 10.1128/JVI.46.2.620-625.1983.

Complementation of a tsa mutant and replication of a recombinant DNA carrying the viral ori region in mouse cells transformed by polyoma virus.

Virology. 1982 Oct 30;122(2):306-17. doi: 10.1016/0042-6822(82)90230-6.

Construction and characterization of viable deletion mutants of simian virus 40 lacking sequences near the 3' end of the early region.

J Virol. 1982 Aug;43(2):489-502. doi: 10.1128/JVI.43.2.489-502.1982.

The transformation of cell growth and transmogrification of DNA synthesis by simian virus 40.

Adv Cancer Res. 1981;34:1-68. doi: 10.1016/s0065-230x(08)60238-9.

Host-virus interactions critical for cellular transformation by polyoma virus and SV40.

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 1,:333-42. doi: 10.1101/sqb.1980.044.01.038.

Separation of lytic and transforming functions of the simian virus 40 A region: two mutants which are temperature sensitive for lytic functions have opposite effects on transformation.

J Virol. 1981 May;38(2):518-28. doi: 10.1128/JVI.38.2.518-528.1981.

Structure and function of the transforming genes of human adenoviruses and SV40.

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 1,:383-99. doi: 10.1101/sqb.1980.044.01.043.

引用本文的文献

A structural rationale for SV40 Vp1 temperature-sensitive mutants and their complementation.

Protein Sci. 2006 Sep;15(9):2207-13. doi: 10.1110/ps.062195606. Epub 2006 Aug 1.

Clinical mutations in the L1 neural cell adhesion molecule affect cell-surface expression.

J Neurosci. 2000 Aug 1;20(15):5696-702. doi: 10.1523/JNEUROSCI.20-15-05696.2000.

Characterization of human papillomavirus type 11 E1 and E2 proteins expressed in insect cells.

J Virol. 1993 May;67(5):2655-63. doi: 10.1128/JVI.67.5.2655-2663.1993.

Simian virus 40 large T antigen host range domain functions in virion assembly.

J Virol. 1994 Jul;68(7):4227-40. doi: 10.1128/JVI.68.7.4227-4240.1994.

Sequence-specific binding of simian virus 40 A protein to nonorigin and cellular DNA.

Mol Cell Biol. 1984 Dec;4(12):2631-8. doi: 10.1128/mcb.4.12.2631-2638.1984.

Simian virus 40 large T-antigen point mutants that are defective in viral DNA replication but competent in oncogenic transformation.

Mol Cell Biol. 1984 Jun;4(6):1125-33. doi: 10.1128/mcb.4.6.1125-1133.1984.

Subcellular distribution of viral structural proteins during simian virus 40 infection.

J Virol. 1984 May;50(2):363-71. doi: 10.1128/JVI.50.2.363-371.1984.

DNA-binding activity of simian virus 40 large T antigen correlates with a distinct phosphorylation state.

J Virol. 1984 Apr;50(1):1-12. doi: 10.1128/JVI.50.1.1-12.1984.

Large-T-antigen-p53 complex formation is not cold sensitive in a cold-sensitive transformant induced by simian virus 40 mutant tsA1499.

J Virol. 1984 Mar;49(3):997-1001. doi: 10.1128/JVI.49.3.997-1001.1984.

Oligomerization of simian virus 40 large T antigen is not necessarily repressed by temperature-sensitive A gene lesions.

J Virol. 1984 Mar;49(3):658-64. doi: 10.1128/JVI.49.3.658-664.1984.

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Characterization of a temperature-sensitive, DNA-positive, nontransforming mutant of simian virus 40.

作者信息

Cosman D J, Tevethia M J

出版信息

Virology. 1981 Jul 30;112(2):605-24. doi: 10.1016/0042-6822(81)90306-8.

DOI:10.1016/0042-6822(81)90306-8

PMID:6266143

Abstract

摘要

相似文献

Characterization of a temperature-sensitive, DNA-positive, nontransforming mutant of simian virus 40.

Virology. 1981 Jul 30;112(2):605-24. doi: 10.1016/0042-6822(81)90306-8.

Mutant carrying deletions in the two simian virus 40 early genes.

J Virol. 1981 Dec;40(3):625-34. doi: 10.1128/JVI.40.3.625-634.1981.

Viral and cellular control of the SV40-transformed phenotype.

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 1,:343-52. doi: 10.1101/sqb.1980.044.01.039.

A simian virus 40 dl884/tsA58 double mutant is temperature sensitive for abortive transformation.

J Virol. 1983 May;46(2):620-5. doi: 10.1128/JVI.46.2.620-625.1983.

Complementation of a tsa mutant and replication of a recombinant DNA carrying the viral ori region in mouse cells transformed by polyoma virus.

Virology. 1982 Oct 30;122(2):306-17. doi: 10.1016/0042-6822(82)90230-6.

Construction and characterization of viable deletion mutants of simian virus 40 lacking sequences near the 3' end of the early region.

J Virol. 1982 Aug;43(2):489-502. doi: 10.1128/JVI.43.2.489-502.1982.

The transformation of cell growth and transmogrification of DNA synthesis by simian virus 40.

Adv Cancer Res. 1981;34:1-68. doi: 10.1016/s0065-230x(08)60238-9.

Host-virus interactions critical for cellular transformation by polyoma virus and SV40.

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 1,:333-42. doi: 10.1101/sqb.1980.044.01.038.

Separation of lytic and transforming functions of the simian virus 40 A region: two mutants which are temperature sensitive for lytic functions have opposite effects on transformation.

J Virol. 1981 May;38(2):518-28. doi: 10.1128/JVI.38.2.518-528.1981.

Structure and function of the transforming genes of human adenoviruses and SV40.

Cold Spring Harb Symp Quant Biol. 1980;44 Pt 1,:383-99. doi: 10.1101/sqb.1980.044.01.043.

引用本文的文献

A structural rationale for SV40 Vp1 temperature-sensitive mutants and their complementation.

Protein Sci. 2006 Sep;15(9):2207-13. doi: 10.1110/ps.062195606. Epub 2006 Aug 1.

Clinical mutations in the L1 neural cell adhesion molecule affect cell-surface expression.

J Neurosci. 2000 Aug 1;20(15):5696-702. doi: 10.1523/JNEUROSCI.20-15-05696.2000.

Characterization of human papillomavirus type 11 E1 and E2 proteins expressed in insect cells.

J Virol. 1993 May;67(5):2655-63. doi: 10.1128/JVI.67.5.2655-2663.1993.

Simian virus 40 large T antigen host range domain functions in virion assembly.

J Virol. 1994 Jul;68(7):4227-40. doi: 10.1128/JVI.68.7.4227-4240.1994.

Sequence-specific binding of simian virus 40 A protein to nonorigin and cellular DNA.

Mol Cell Biol. 1984 Dec;4(12):2631-8. doi: 10.1128/mcb.4.12.2631-2638.1984.

Simian virus 40 large T-antigen point mutants that are defective in viral DNA replication but competent in oncogenic transformation.

Mol Cell Biol. 1984 Jun;4(6):1125-33. doi: 10.1128/mcb.4.6.1125-1133.1984.

Subcellular distribution of viral structural proteins during simian virus 40 infection.

J Virol. 1984 May;50(2):363-71. doi: 10.1128/JVI.50.2.363-371.1984.

DNA-binding activity of simian virus 40 large T antigen correlates with a distinct phosphorylation state.

J Virol. 1984 Apr;50(1):1-12. doi: 10.1128/JVI.50.1.1-12.1984.

Large-T-antigen-p53 complex formation is not cold sensitive in a cold-sensitive transformant induced by simian virus 40 mutant tsA1499.

J Virol. 1984 Mar;49(3):997-1001. doi: 10.1128/JVI.49.3.997-1001.1984.

Oligomerization of simian virus 40 large T antigen is not necessarily repressed by temperature-sensitive A gene lesions.

J Virol. 1984 Mar;49(3):658-64. doi: 10.1128/JVI.49.3.658-664.1984.