Vesin P, Belaiche J, Cattan D
Sem Hop. 1981;57(21-24):1112-4.
A study of renal tubular acidification in two cirrhotic patients with functional renal failure and partly compensated metabolic acidosis. Unlike renal distal tubular acidosis, but alike what is seen in organic renal failure, renal acidification was nearly normal and could not account per se for the development of metabolic acidosis. The multiple cause of the association in the cirrhotic of hyperchloremia and hypobicarbonatemia are reviewed (respiratory alkalosis, metabolic acidosis due to renal failure, distal tubular acidosis) and the diagnostic procedures outlined.
对两名患有功能性肾衰竭和部分代偿性代谢性酸中毒的肝硬化患者肾小管酸化的研究。与肾性远端肾小管酸中毒不同，但与有机性肾衰竭所见相似，肾小管酸化几乎正常，本身不能解释代谢性酸中毒的发生。本文回顾了肝硬化患者高氯血症和低碳酸氢盐血症相关的多种原因（呼吸性碱中毒、肾衰竭所致代谢性酸中毒、远端肾小管酸中毒）并概述了诊断程序。
