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Glial contribution to seizure: K+ activation of (Na+, K+)-ATPase in bulk isolated glial cells and synaptosomes of epileptogenic cortex.

作者信息

Grisar T, Franck G, Delgado-Escueta A V

出版信息

Brain Res. 1983 Feb 14;261(1):75-84. doi: 10.1016/0006-8993(83)91285-4.

DOI:10.1016/0006-8993(83)91285-4
PMID:6301630
Abstract
摘要

相似文献

1
Glial contribution to seizure: K+ activation of (Na+, K+)-ATPase in bulk isolated glial cells and synaptosomes of epileptogenic cortex.
Brain Res. 1983 Feb 14;261(1):75-84. doi: 10.1016/0006-8993(83)91285-4.
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Milacemide stimulates deficient glial Na+, K(+)-ATPase in freezing-induced epileptogenic cortex of cats.
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Na+ K+-ATPase within neurons and glia in the generation of seizures.神经元和神经胶质细胞中的钠钾ATP酶与癫痫发作的产生。
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Na+-K+-ATPase activity of glial, neuronal, and synaptosomal enriched fractions from normal and freezing-injured rabbit cerebral cortex.正常和冷冻损伤兔大脑皮层的神经胶质、神经元及富含突触体组分的钠钾ATP酶活性
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Astroglial contribution in human temporal lobe epilepsy: K+ activation of Na+,K+-ATPase in bulk isolated glial cells and synaptosomes.
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Managing Brain Extracellular K(+) during Neuronal Activity: The Physiological Role of the Na(+)/K(+)-ATPase Subunit Isoforms.神经元活动期间对脑胞外钾离子的调控:钠钾ATP酶亚基异构体的生理作用
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Na+-K+-ATPase activity of glial, neuronal, and synaptosomal enriched fractions from normal and freezing-injured rabbit cerebral cortex.正常和冷冻损伤兔大脑皮层的神经胶质、神经元及富含突触体组分的钠钾ATP酶活性
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