[Alteration of energy-dependent functions of liver mitochondria during the interaction of the carrier of adenine nucleotides with palmitoyl-CoA].

Palmitoyl-CoA, at the concentrations which did cause uncoupling of oxidative phosphorylation, decreased the energy pool of liver mitochondria in presence of oligomycine. Characteristic ability of palmitoyl-CoA to increase the proton transport in mitochondrial membranes was responsible for the deenergizing action.