Mitchel R E, Morrison D P
Radiat Res. 1983 Oct;96(1):95-9.
When exponentially growing diploid wild type Saccharomyces cerevisiae cells were subjected to a sudden rise in temperature (heat shock) they responded by increasing their resistance to the lethal effects of ultraviolet light. We have previously reported heat shock-induced increases in heat and ionizing radiation resistance. The shock-induced rise in resistance to uv light reported here was examined in terms of DNA repair capacity, and we find that the increase is due to induction of the recombinational repair system with no significant response from the uv-excision repair process.
当指数生长的二倍体野生型酿酒酵母细胞受到温度突然升高(热休克)的影响时,它们会通过增强对紫外线致死效应的抗性来做出反应。我们之前曾报道过热休克诱导热抗性和抗电离辐射能力的增强。本文所报道的热休克诱导的对紫外线抗性的提高,我们从DNA修复能力方面进行了研究,并且发现这种提高是由于重组修复系统的诱导,而紫外线切除修复过程没有显著反应。