Saceda M, García-Morales P, Mato J M, Malaisse W J, Valverde I
Biochem Int. 1984 Mar;8(3):445-52.
Glucose provokes a transient stimulation of phospholipid methylation in rat pancreatic islets, possibly by increasing phospholipid methyltransferase activity. The association of DL-homocysteine and 3-deazaadenosine inhibits phospholipid methylation. The methylation of phospholipids may play a role in the stimulus-secretion coupling for glucose-induced insulin release.