Henson G L, Dawes G S, Redman C W
Br J Obstet Gynaecol. 1983 Jun;90(6):516-21. doi: 10.1111/j.1471-0528.1983.tb08958.x.
A quantitative comparison was made of fetal heart-rate (FHR) traces obtained shortly before delivery, and of umbilical artery blood gas values, in 49 women delivered by caesarean section for reasons other than fetal compromise and in 23 women delivered by section for antenatal fetal compromise judged from visual inspection of conventional FHR recordings. The FHR traces in the compromised group of fetuses were abnormal, with low variability and a high incidence of decelerations, but there was no significant difference from the normal group in metabolic acidaemia, as judged by the base excess of umbilical artery blood. The compromised group consisted almost exclusively of infants small-for-gestational age. These results suggest that the FHR pattern of the compromised fetuses is not the direct result of intrauterine asphyxia but of some other factor associated with growth retardation.
对49例因胎儿窘迫以外的原因行剖宫产分娩的妇女以及23例根据传统胎儿心率（FHR）记录的目视检查判断为产前胎儿窘迫而行剖宫产分娩的妇女，在分娩前不久获得的胎儿心率（FHR）轨迹以及脐动脉血气值进行了定量比较。胎儿窘迫组的FHR轨迹异常，变异性低且减速发生率高，但根据脐动脉血碱剩余判断，其代谢性酸血症与正常组无显著差异。窘迫组几乎全部为小于胎龄儿。这些结果表明，窘迫胎儿的FHR模式不是宫内窒息的直接结果，而是与生长迟缓相关的其他因素所致。
