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Inflammatory mediators released by complement-derived peptides.

作者信息

Bult H, Herman A G

出版信息

Agents Actions. 1983 Aug;13(5-6):405-14. doi: 10.1007/BF02176403.

DOI:10.1007/BF02176403
PMID:6416038
Abstract
摘要

相似文献

1
Inflammatory mediators released by complement-derived peptides.补体衍生肽释放的炎症介质。
Agents Actions. 1983 Aug;13(5-6):405-14. doi: 10.1007/BF02176403.
2
Role of complement anaphylatoxins in neutrophil arachidonic acid metabolism.补体过敏毒素在中性粒细胞花生四烯酸代谢中的作用。
Prog Clin Biol Res. 1985;199:173-84.
3
Complement components in relation to macrophage function.与巨噬细胞功能相关的补体成分。
Agents Actions. 1983 Aug;13(5-6):415-28. doi: 10.1007/BF02176404.
4
Complement as a mediator of granulocyte adherence and margination: studies based on the acute neutropenia of filtration leukapheresis.补体作为粒细胞黏附和靠边化的介质：基于过滤性白细胞单采术所致急性中性粒细胞减少症的研究
Prog Clin Biol Res. 1977;13:243-58.
5
Contribution of complement factors and reaction pathways to inflammation.补体因子和反应途径在炎症中的作用。
Agents Actions Suppl. 1982;11:159-78.
6
Vascular permeability changes induced by complement-derived peptides.补体衍生肽诱导的血管通透性变化
Agents Actions. 1983 Aug;13(5-6):451-5. doi: 10.1007/BF02176415.
7
Absence of induction of IL-1 production in human monocytes by complement fragments.补体片段不能诱导人单核细胞产生白细胞介素-1。
J Immunol. 1989 Jan 1;142(1):173-8.
8
C3a-induced lysosomal enzyme secretion from human neutrophils: lack of inhibition by f met-leu-phe antagonists and inhibition by arachidonic acid antagonists.C3a诱导人中性粒细胞溶酶体酶分泌：甲酰甲硫氨酸-亮氨酸-苯丙氨酸拮抗剂无抑制作用，花生四烯酸拮抗剂有抑制作用。
Int Arch Allergy Appl Immunol. 1982;67(3):227-32. doi: 10.1159/000233023.
9
Role of the complement anaphylatoxins in inflammation and hypersensitivity reactions in the lung.补体过敏毒素在肺部炎症和超敏反应中的作用。
Surv Synth Pathol Res. 1985;4(5-6):423-42. doi: 10.1159/000156994.
10
Role of complements C3 and C5 in the phagocytosis of liposomes by human neutrophils.补体C3和C5在人中性粒细胞吞噬脂质体中的作用。
Pharm Res. 1991 Jan;8(1):65-9. doi: 10.1023/a:1015830306839.

引用本文的文献

1
A low molecular weight proinflammatory factor from rat spleen lymphocytes. Isolation and partial characterization.一种来自大鼠脾脏淋巴细胞的低分子量促炎因子。分离及部分特性鉴定。
Inflammation. 2007 Aug;30(3-4):87-96. doi: 10.1007/s10753-007-9024-4. Epub 2007 May 2.
2
The modulation of peritoneal macrophage chemiluminescence by acute pleural inflammation, prostanoids and cyclo/lipoxygenase inhibitors.急性胸膜炎、前列腺素及环氧化酶/脂氧化酶抑制剂对腹膜巨噬细胞化学发光的调节作用
Agents Actions. 1985 Dec;17(2):184-91. doi: 10.1007/BF01966590.
3
In vitro inhibition of the classical and alternate pathways of activation of human complement by N acetyl aspartyl glutamic acid (NAAGA).

本文引用的文献

1
Control of vascular permeability by polymorphonuclear leukocytes in inflammation.炎症中多形核白细胞对血管通透性的控制
Nature. 1981 Feb 19;289(5799):646-50. doi: 10.1038/289646a0.
2
C5a-induced tracheal contraction: effect of an SRS-A antagonist and inhibitors of arachidonate metabolism.C5a诱导的气管收缩：SRS-A拮抗剂和花生四烯酸代谢抑制剂的作用
J Immunol. 1981 Jan;126(1):313-6.
3
The regulation of human eosinophil function by endogenous mono-hydroxy-eicosatetraenoic acids (HETEs).内源性单羟基二十碳四烯酸（HETEs）对人类嗜酸性粒细胞功能的调节。
N-乙酰天冬氨酰谷氨酸（NAAGA）对人补体经典激活途径和替代激活途径的体外抑制作用
Agents Actions. 1988 Jun;24(1-2):137-44. doi: 10.1007/BF01968092.
J Immunol. 1980 Feb;124(2):926-33.
4
Human complement in the arachidonic acid transformation pathway in platelets.血小板中花生四烯酸转化途径中的人类补体。
J Exp Med. 1981 Feb 1;153(2):257-68. doi: 10.1084/jem.153.2.257.
5
Neutropenia induced by platelet-activating factor (PAF-acether) released from neutrophils: the inhibitory effect of prostacyclin (PGI2).中性粒细胞释放的血小板活化因子（PAF-乙醚）诱导的中性粒细胞减少：前列环素（PGI2）的抑制作用。
Agents Actions. 1981 Dec;11(6-7):550-3. doi: 10.1007/BF01978735.
6
Effect of complement activation on the biosynthesis of prostacyclin (PGI2) by rabbit peritoneum in vitro.补体激活对兔腹膜体外合成前列环素（PGI2）的影响。
Arch Int Pharmacodyn Ther. 1981 Oct;253(2):327-9.
7
The proteolytic activation systems of complement.补体的蛋白水解激活系统。
Annu Rev Biochem. 1981;50:433-64. doi: 10.1146/annurev.bi.50.070181.002245.
8
Blood levels of 6-oxo-prostaglandin F 1 alpha during endotoxin-induced hypotension in rabbits.兔子内毒素诱导低血压期间6-氧代前列腺素F1α的血药浓度
Eur J Pharmacol. 1980 Apr 11;63(1):47-56. doi: 10.1016/0014-2999(80)90115-6.
9
C3a induced activation and stimulus specific reversible desensitization of guinea pig platelets.C3a诱导豚鼠血小板活化及刺激特异性可逆脱敏。
Agents Actions Suppl. 1982;11:147-57.
10
Anaphylactic actions of platelet-activating factor.血小板活化因子的过敏反应作用。
Am J Pathol. 1981 Oct;105(1):64-9.