Goncharova V A
Vopr Med Khim. 1983 Nov-Dec;29(6):34-9.
Metabolism of catecholamines and serotonin was studied in experiments with ischemia of respiratory tissue and hypoxic hypoxia, in cases of acute and chronic inflammatory bronchopulmonary processes as well as in lung homogenates obtained from patients with bronchiectases. Long-term effects of ischemia, hypoxia and chronic inflammatory bronchopulmonary process caused inhibition of oxidative deamination of monoamines, as a result of which the barrier function of lung might be impaired.
在呼吸组织缺血和低氧性缺氧的实验中，以及在急性和慢性炎症性支气管肺疾病的病例中，还有从支气管扩张症患者获取的肺匀浆中，对儿茶酚胺和5-羟色胺的代谢进行了研究。缺血、缺氧和慢性炎症性支气管肺疾病的长期影响导致单胺氧化脱氨基作用受到抑制，其结果可能是肺的屏障功能受损。
