Bonnheim D C, Petrelli N J, Sternberg A, Mittelman A
J Surg Oncol. 1984 Jul;26(3):172-5. doi: 10.1002/jso.2930260307.
Supravesical urinary diversion using a jejunal conduit may be associated with hyponatremia, hypochloremic-acidosis, hyperkalemia, azotemia, and a clinical picture of nausea, vomiting, dehydration, muscular weakness, elevated temperature, and lethargy. This syndrome is secondary to the loss of sodium chloride into the urine passing through the conduit and absorption of potassium and urea from it. Treatment and prevention of this syndrome consist of adequate supplements of sodium chloride and hydration. Intravenous hyperalimentation as the precipitating factor of a severe form of this syndrome and its successful management are described. The pathophysiology of the jejunal conduit syndrome is also discussed. Great selectivity and extreme caution are recommended with respect to the use of intravenous hyperalimentation in patients with jejunal conduits.
使用空肠导管进行膀胱上尿路改道可能与低钠血症、低氯性酸中毒、高钾血症、氮质血症以及恶心、呕吐、脱水、肌肉无力、体温升高和嗜睡的临床表现相关。该综合征继发于氯化钠进入通过导管的尿液中丢失以及钾和尿素从中吸收。该综合征的治疗和预防包括充分补充氯化钠和补液。描述了静脉高营养作为该综合征严重形式的诱发因素及其成功处理。还讨论了空肠导管综合征的病理生理学。对于有空肠导管的患者，建议在使用静脉高营养时要高度谨慎。
