Práger P, Strenger J, Puppi A, Angyal T, Detre Z, Matkovics B
Acta Physiol Hung. 1984;64(3-4):489-94.
It was found that alcohol caused pathological and irreversible decrements of the redox-state potential (redosis) in the pancreas and the liver. On giving reducing agents, a compensatory oxidosis developed, and this could prevent the cell damaging effect of alcohol. This effect was most marked in the reversible phase, while it could be effective also in the irreversible phase. By measuring the redox buffer capacity in the tissues it is possible to determine the measure of reversibility of the injury caused by alcohol.
研究发现，酒精会导致胰腺和肝脏的氧化还原状态电位（氧化还原病）出现病理性且不可逆的降低。给予还原剂时，会出现代偿性氧化病，这可以预防酒精对细胞的损伤作用。这种作用在可逆阶段最为显著，而在不可逆阶段也可能有效。通过测量组织中的氧化还原缓冲能力，可以确定酒精所致损伤的可逆程度。
