Tank E S, Alexander S R, Craven R M
J Urol. 1980 Nov;124(5):692-4. doi: 10.1016/s0022-5347(17)55613-x.
We diagnosed and treated 5 children with huge bladders and hydroureteronephrosis secondary to polyuria, resulting from either familial nephrogenic diabetes insipidus, medullary cystic disease, central diabetes insipidus secondary to panhypopituitarism or psychogenic polydipsia. Polyuria was documented by the measurement of 24-hour urine volumes. The possibility of anatomic and physiologic outlet obstruction was eliminated by flowmetry, voiding cystourethrography and endoscopy.
我们诊断并治疗了5例因多尿继发巨大膀胱及输尿管肾积水的儿童，多尿的病因包括家族性肾性尿崩症、髓质囊性疾病、全垂体功能减退继发的中枢性尿崩症或精神性烦渴。通过测量24小时尿量记录多尿情况。通过尿流率测定、排尿性膀胱尿道造影和内镜检查排除了解剖学和生理学出口梗阻的可能性。
