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遗传性下丘脑性尿崩症大鼠的球旁器

The juxtaglomerular apparatus of rats with hereditary hypothalamic diabetes insipidus.

作者信息

Peter S, Möhring J

出版信息

Cell Tissue Res. 1978 Apr 17;188(2):335-9. doi: 10.1007/BF00222642.

DOI:10.1007/BF00222642
PMID:647755
Abstract

The juxtaglomerular apparatus (JGA) of rats with hereditary hypothalamic diabetes insipidus (DI) was studied. Plasma concentration of renin and angiotensin II, as well as serum sodium concentration and serum osmolality of DI rats are elevated. The morphological examination reveals no characteristic alteration of the epitheloid cells. The results show that the epitheloid cells are sufficiently adapted for the higher release of renin.

摘要

对患有遗传性下丘脑性尿崩症(DI)的大鼠的球旁器(JGA)进行了研究。DI大鼠的血浆肾素和血管紧张素II浓度以及血清钠浓度和血清渗透压均升高。形态学检查显示上皮样细胞无特征性改变。结果表明,上皮样细胞足以适应肾素的更高释放。

相似文献

1
The juxtaglomerular apparatus of rats with hereditary hypothalamic diabetes insipidus.遗传性下丘脑性尿崩症大鼠的球旁器
Cell Tissue Res. 1978 Apr 17;188(2):335-9. doi: 10.1007/BF00222642.
2
Discrepancy between aldosterone production and renin-angiotensin system activity in Brattleboro rats.布拉特伯勒大鼠醛固酮生成与肾素-血管紧张素系统活性之间的差异。
Acta Physiol Pharmacol Bulg. 1987;13(4):43-50.
3
Coexistence of renin and angiotensin II in epitheloid cell secretory granules of rat kidney.肾素与血管紧张素II在大鼠肾脏上皮样细胞分泌颗粒中的共存。
Histochemistry. 1984;81(1):39-45. doi: 10.1007/BF00495399.
4
[Dynamics of epithelioid cells in juxtaglomerular apparatus].[肾小球旁器中上皮样细胞的动态变化]
Verh Anat Ges. 1976(70 Pt 1):111-9.
5
Morphological correlates of renin and aldosterone secretion in the Brattleboro rat.布拉特洛维大鼠肾素和醛固酮分泌的形态学关联
Ann N Y Acad Sci. 1982;394:291-8. doi: 10.1111/j.1749-6632.1982.tb37440.x.
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The renin and isorenin-angiotensin system in rats with hereditary hypothalamic diabetes insipidus.遗传性下丘脑性尿崩症大鼠的肾素及异肾素-血管紧张素系统
Neuropharmacology. 1978 Nov;17(11):919-23. doi: 10.1016/0028-3908(78)90132-6.
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Effects of physical training during the growth on the granulation of myo-epithelioid cells of the rat kidney.
Acta Anat (Basel). 1991;141(4):329-34. doi: 10.1159/000147144.
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Potassium deficiency in rats with hereditary diabetes insipidus.遗传性尿崩症大鼠的钾缺乏症
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The renal kallikrein-kinin system in Brattleboro rats with hereditary hypothalamic diabetes insipidus. Missing relationship between antidiuretic hormone and the renal kallikrein-kinin system.遗传性下丘脑性尿崩症的布拉特洛伯大鼠的肾激肽释放酶-激肽系统。抗利尿激素与肾激肽释放酶-激肽系统之间缺乏关联。
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Am J Pathol. 1984 May;115(2):212-24.

本文引用的文献

1
THE ASSAY OF RENIN IN SINGLE GLOMERULI IN THE NORMAL RABBIT AND THE APPEARANCE OF THE JUXTAGLOMERULAR APPARATUS.正常家兔单个肾小球中肾素的测定及球旁器的出现
J Physiol. 1965 Feb;176(3):418-28. doi: 10.1113/jphysiol.1965.sp007559.
2
FAMILIAL HYPOTHALAMIC DIABETES INSIPIDUS IN RATS (BRATTLEBORO STRAIN).大鼠(布拉德福德品系)的家族性下丘脑性尿崩症
Am J Physiol. 1964 Feb;206:425-30. doi: 10.1152/ajplegacy.1964.206.2.425.
3
The use of lead citrate at high pH as an electron-opaque stain in electron microscopy.在电子显微镜检查中,将高pH值的柠檬酸铅用作电子不透明染色剂。
J Cell Biol. 1963 Apr;17(1):208-12. doi: 10.1083/jcb.17.1.208.
4
Hereditary hypothalamic diabetes insipidus in rats (Brattleboro strain). A useful experimental model.大鼠遗传性下丘脑性尿崩症(布拉德福德品系)。一种有用的实验模型。
Am J Med. 1967 May;42(5):814-27. doi: 10.1016/0002-9343(67)90098-8.
5
Inhibition of renin release in the dog by vasopressin and vasotocin.血管加压素和催产素对犬肾素释放的抑制作用。
Circ Res. 1968 Nov;23(5):605-9. doi: 10.1161/01.res.23.5.605.
6
Control of renin release.肾素释放的调控
Physiol Rev. 1967 Jul;47(3):359-82. doi: 10.1152/physrev.1967.47.3.359.
7
Studies on the juxtaglomerular apparatus. II. Quantitative morphology after adrenalectomy.肾小球旁器的研究。II. 肾上腺切除术后的定量形态学
Cell Tissue Res. 1974;151(4):457-69. doi: 10.1007/BF00222991.
8
Potassium deficiency in rats with hereditary diabetes insipidus.遗传性尿崩症大鼠的钾缺乏症
Am J Physiol. 1974 Oct;227(4):916-20. doi: 10.1152/ajplegacy.1974.227.4.916.
9
Studies on the juxtaglomerular apparatus. III. Quantitative morphology after treatment with deoxycorticosterone (DOC).肾小球旁器的研究。III. 脱氧皮质酮(DOC)治疗后的定量形态学
Cell Tissue Res. 1974;151(4):471-80. doi: 10.1007/BF00222992.
10
[The juxtaglomerular apparatus of the kidney in diabetes insipidus (author's transl)].尿崩症时肾脏的球旁器(作者译)
Beitr Pathol. 1974 Apr;151(4):361-72.