[实验性心肌梗死中的脂质过氧化：高压氧疗的作用]
[Lipid peroxidation in experimental myocardial infarct: the action of hyperbaric oxygenation].
作者信息
Meerson F Z, Didenko V V, Savov V M, Kopaladze R A, Dosmagambetova R S
出版信息
Biull Eksp Biol Med. 1984 Oct;98(10):398-400.
PMID:6498314
Abstract
Rats with experimental myocardial infarction demonstrated decrease in the activity of superoxide dismutase and catalase and increase in the content of lipid peroxidation (LPO) products and Schiff bases both in and outside the area of necrosis. The combined ischemic damage and hyperbaric oxygenation resulted in the over additive effect of accumulation of LPO products in and outside the area of infarction. The data suggest that it is desirable to use antioxidants during hyperbaric oxygenation.
摘要
实验性心肌梗死大鼠坏死区内外超氧化物歧化酶和过氧化氢酶活性降低，脂质过氧化（LPO）产物和席夫碱含量增加。缺血损伤与高压氧合联合作用导致梗死区内外LPO产物积累产生超相加效应。这些数据表明，在高压氧合过程中使用抗氧化剂是可取的。
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