Belhassen B, Caspi A, Miller H, Shapira I, Laniado S
J Am Coll Cardiol. 1984 Dec;4(6):1302-6. doi: 10.1016/s0735-1097(84)80153-9.
A 38 year old man with arrhythmogenic right ventricular dysplasia underwent electrophysiologic study with extensive endocardial mapping of the right ventricle. During sinus rhythm, markedly reduced amplitude with or without prolonged duration was noted in multiple electrograms of the right ventricle. Delayed activation of the right ventricular outflow tract electrogram was present. Two clinical types of ventricular tachycardia with a left bundle branch block configuration and either an inferior or a superior axis were induced by ventricular stimulation. Endocardial activation sequence during the tachycardias suggested that the tachycardia with an inferior axis originated near the infundibulum, while that with a superior axis originated near the midinferior area of the right ventricle. Both areas have been previously shown to be frequently impaired in right ventricular dysplasia.
一名患有致心律失常性右心室发育不良的38岁男性接受了电生理研究，对右心室进行了广泛的心内膜标测。在窦性心律期间，右心室多个电图显示振幅明显降低，持续时间可延长或不延长。右心室流出道电图存在延迟激活。通过心室刺激诱发了两种临床类型的室性心动过速，呈左束支传导阻滞形态，电轴向下或向上。心动过速期间的心内膜激动顺序表明，电轴向下的心动过速起源于漏斗部附近，而电轴向上的心动过速起源于右心室中下区域附近。先前已表明这两个区域在右心室发育不良中经常受损。
