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[足内翻畸形偏瘫患者腿部肌肉的肌电图分析]

[Electromyographic analysis of the leg muscles in the hemiplegic patients with equinovarus deformity of the foot].

作者信息

Iwaya T

出版信息

Nihon Seikeigeka Gakkai Zasshi. 1984 Oct;58(10):961-73.

PMID:6520494
Abstract

A series of EMG study of the leg muscles was carried out with a wire electrode in 86 hemiplegic patients of stroke to visualize the role of each muscle either in the development of equinovarus deformity of the foot or in correcting the deformity through tendon transfer. The muscles examined were anterior tibialis, posterior tibialis, gastrocnemius, soleus, flexor digitorum longus and peroneus brevis of the affected side. Tonic discharge of those muscles was recorded as the patients were elevating the affected leg in supine, sitting or standing posture or were standing on legs. On lifting up the affected limb, most patients showed electrical activity of anterior tibialis with or without simultaneous activity of other muscles, most frequently with that of flexor digitorum longus or gastrocnemius. When the patient stood on legs electromyographic discharge appeared most frequently in soleus. Varus deformity of the foot significantly correlated to the lack of the electrical activity of peroneus brevis. Both such abnormal activity of anterior tibialis and the lack of activity of peroneus seemed to be the main causes for the varus deformity. Postoperative EMG study in the patients who underwent Watkins-Barr procedure of anterior transfer of the posterior tibialis tendon, showed that the posterior tibialis was rather inactive both in elevating the leg and in standing on legs. Varus deformity was corrected independent of the discharge of posterior tibialis. The author concluded that the correction of the varus deformity after Watkins-Barr procedure was mainly obtained from the tenodesis effect. The tenodesis provides the checkline effect on the equinus and varus deformity, which reinforces the dorsiflexing action of anterior tibialis and attenuates its inverting action of the same muscle.

摘要

对86例中风偏瘫患者使用线状电极进行了一系列腿部肌肉的肌电图研究,以观察每块肌肉在足内翻畸形发展过程中的作用,以及通过肌腱转移矫正畸形时的作用。所检查的肌肉为患侧的胫前肌、胫后肌、腓肠肌、比目鱼肌、趾长屈肌和腓骨短肌。当患者以仰卧、坐姿或站姿抬高患侧下肢,或站立时,记录这些肌肉的张力性放电情况。抬起患侧肢体时,大多数患者显示胫前肌有电活动,其他肌肉可同时或不同时活动,最常见的是与趾长屈肌或腓肠肌同时活动。患者站立时,肌电图放电最常出现在比目鱼肌。足内翻畸形与腓骨短肌电活动缺失显著相关。胫前肌的这种异常活动和腓骨肌活动缺失似乎都是内翻畸形的主要原因。对接受胫后肌腱前移位的Watkins-Barr手术患者进行术后肌电图研究发现,胫后肌在抬腿和站立时均相当不活跃。内翻畸形的矫正与胫后肌的放电无关。作者得出结论,Watkins-Barr手术后内翻畸形的矫正主要源于腱固定效应。腱固定对马蹄足和内翻畸形起到了制动作用,增强了胫前肌的背屈作用,并减弱了该肌肉的内翻作用。

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