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对钙拮抗剂敏感的束支性心动过速

Fascicular tachycardia sensitive to calcium antagonists.

作者信息

Ward D E, Nathan A W, Camm A J

出版信息

Eur Heart J. 1984 Nov;5(11):896-905. doi: 10.1093/oxfordjournals.eurheartj.a061589.

Abstract

Five patients with recurrent tachycardias exhibiting right bundle branch block with left axis deviation were referred for investigation. In each case, a supraventricular mechanism was suspected. During sinus rhythm the QRS morphology and axis (-10 to +60 degrees) and HV intervals were normal. Tachycardia was initiated by timed ventricular premature stimuli in 4 patients, rapid ventricular pacing in 3 patients and rapid atrial pacing in 2 patients. The tachycardia cycle length varied from 275 to 380 ms with right bundle branch block and a leftward axis change of 30 to 125 degrees at the onset of the tachycardia. The HV interval ranged from +15 to -20 ms. In each patient ventriculoatrial dissociation occurred spontaneously or could be induced. All tachycardia could be terminated or greatly slowed by calcium antagonists. These data are consistent with an unusual reentrant mechanism of tachycardia located in the posterior fascicle of the left bundle branch.

摘要

5例反复出现心动过速且表现为右束支传导阻滞伴左前分支阻滞的患者被转诊进行检查。每例患者均怀疑为室上性机制。在窦性心律时,QRS波形态、电轴(-10至+60度)及HV间期均正常。4例患者通过适时的室性早搏刺激诱发心动过速,3例患者通过快速心室起搏诱发,2例患者通过快速心房起搏诱发。心动过速周期长度在275至380毫秒之间,心动过速发作时伴有右束支传导阻滞及电轴向左改变30至125度。HV间期在+15至-20毫秒之间。每例患者均自发出现或可诱发室房分离。所有心动过速均可被钙拮抗剂终止或显著减慢。这些数据与位于左束支后分支的一种不寻常的折返性心动过速机制相符。

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