Richards W O, Shin B
Crit Care Med. 1984 Mar;12(3):202-3. doi: 10.1097/00003246-198403000-00010.
Polyuria, peaking at 2000 ml/h, was seen in a patient after resuscitation of hemorrhagic shock and a brief period of oliguria. This unusual polyuria appears to be a consequence of renal tubular dysfunction that persisted after glomerular filtration rate had returned to normal.
一名患者在失血性休克复苏后出现多尿,峰值达2000毫升/小时,且有一段短暂的少尿期。这种异常多尿似乎是肾小管功能障碍的结果,在肾小球滤过率恢复正常后该功能障碍仍持续存在。