动作诱发性节律性肌张力障碍：一例尸检病例
Action-induced rhythmic dystonia: an autopsy case.
作者信息
Sunohara N, Mukoyama M, Mano Y, Satoyoshi E
出版信息
Neurology. 1984 Mar;34(3):321-7. doi: 10.1212/wnl.34.3.321.
PMID:6608063
Abstract
We studied a patient with action-induced rhythmic dystonia that followed a stroke. Postmortem studies showed an infarct in the right posterolateral ventral part of the thalamus. Electrophysiologic analysis indicated that the eliciting factor of the involuntary movement was an impulse, promoting voluntary contraction of muscle. CSF 5-HIAA content was low, and HVA was high. Administration of 5-HTP and clonazepam abolished the involuntary movements.
摘要
我们研究了一名中风后出现动作诱发性节律性肌张力障碍的患者。尸检研究显示丘脑右后外侧腹侧部分存在梗死灶。电生理分析表明，非自主运动的诱发因素是一种冲动，它促进了肌肉的自主收缩。脑脊液中5-羟吲哚乙酸（5-HIAA）含量低，高香草酸（HVA）含量高。给予5-羟色氨酸（5-HTP）和氯硝西泮可消除非自主运动。
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