Krnjević K, Lamour Y, MacDonald J F, Nistri A
Can J Physiol Pharmacol. 1978 Jun;56(3):516-20. doi: 10.1139/y78-079.
In cats under Dial anesthesia, microiontophoretic injections of Co2+ or Mn2+ outside lumbosacral motoneurones regularly diminish the postspike hyperpolarizing after-potential (AHP) without any clear reduction of the deplorarizing after-potential (ADP). Comparable injections of Sr2+ have the opposite effect of augmenting the AHP. These results indicate that the AHP is indeed initiated by an influx of calcium linked to the action potential, but the ADP is unlikely to be mainly a manifestation of a calcium inward current.
在处于狄奥麻醉状态的猫身上，在腰骶运动神经元外部微量离子电渗注入Co2+或Mn2+通常会使锋电位后的超极化后电位（AHP）减小，而去极化后电位（ADP）没有明显降低。类似地注入Sr2+则有相反的效果，即增强AHP。这些结果表明，AHP确实是由与动作电位相关的钙内流引发的，但ADP不太可能主要是钙内向电流的表现。
