[Mechanical activity of the system of contractile proteins, energy supply of the myocardium and disruption of calcium transport in inflammatory lesions of the myocardium].

In rabbit experiments it has been shown that in toxico-allergic myocarditis the disordered utilization of ATP energy is responsible for acute insufficiency of the myocardial contractile function, which results in a marked energy deficiency due to mitochondrial damage, the Ca2+ absorbtion and binding by the fragmented sarcoplasmatic reticulum being diminished.