尿毒症神经病变中是否会发生轴突传导的短暂阻断？
Does transient blocking of axonal conduction occur in uraemic neuropathy?
作者信息
Meyer D W, Halonen J P, Lang A H, Kuusela V, Forsström J
出版信息
J Neurol Neurosurg Psychiatry. 1978 Jun;41(6):525-31. doi: 10.1136/jnnp.41.6.525.
Abstract
In uraemic and other patients with neuropathies, motor unit spike intervals were analysed using a special computer program to detect prolonged intervals of twice the normal duration, so-called "double intervals." These may have different causes--trigger failure, double discharge, or a transient fall in the firing rate of coactive motor units. There was not sufficient evidence that transient conduction blockings will occur in the main axon in uraemic neuropathies.
摘要
在尿毒症及其他患有神经病变的患者中，使用一种特殊的计算机程序分析运动单位放电间隔，以检测持续时间为正常两倍的延长间隔，即所谓的“双倍间隔”。这些可能有不同原因——触发失败、双重放电或共同激活的运动单位放电频率短暂下降。没有足够证据表明在尿毒症神经病变中主要轴突会发生短暂性传导阻滞。
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