Rubel L R
Ann Clin Lab Sci. 1984 Mar-Apr;14(2):130-6.
The histopathology of hepatic injury associated with heatstroke is described, with particular attention being devoted to the chronology of histologic alterations. These alterations are discussed with respect to their pathogenesis. Some lesions appear to be a direct result of thermal injury to the liver, while others are secondary effects of clinical risk factors for heatstroke, such as obesity, or reflect systemic complications which include circulatory failure, hemolysis, and endothelial injury.
本文描述了与中暑相关的肝损伤的组织病理学,特别关注组织学改变的时间顺序。并就这些改变的发病机制进行了讨论。一些病变似乎是肝脏热损伤的直接结果,而其他病变则是中暑临床危险因素(如肥胖)的继发效应,或反映了包括循环衰竭、溶血和内皮损伤在内的全身并发症。