Lerman B B, Marchlinski F E, Kempf F C, Buxton A E, Waxman H L, Josephson M E
Int J Cardiol. 1984 Apr;5(4):449-60. doi: 10.1016/0167-5273(84)90080-9.
Intra-Hisian delay is usually associated with fascicular conduction abnormalities. We report our experience in 23 patients who had intra-Hisian delay as their only conduction defect. First-degree intra-Hisian delay (His bundle deflection greater than or equal to 30 msec) was present in 17 patients, 5 of whom also had split His potentials. Three of these patients received permanent pacemakers, all of whom had a history of syncope. Higher degrees of intra-Hisian delay were observed either spontaneously or in response to atrial pacing in 6 patients. Five of these patients were treated with permanent pacing, 4 of whom had symptomatic bradyarrhythmias and/or syncope. No patient has had a recurrence of symptoms following pacemaker insertion during the mean 31-month follow-up. No patient with first-degree intra-Hisian delay not receiving a pacemaker developed symptomatic bradyarrhythmias or syncope during a mean follow-up of 21 months.
希氏束内延迟通常与束支传导异常相关。我们报告了23例仅存在希氏束内延迟这一传导缺陷患者的治疗经验。17例患者存在一度希氏束内延迟（希氏束波时限大于或等于30毫秒），其中5例还存在希氏束电位分裂。这些患者中有3例接受了永久性起搏器植入，均有晕厥病史。6例患者自发或在心房起搏时观察到更高程度的希氏束内延迟。其中5例患者接受了永久性起搏治疗，4例有症状性缓慢性心律失常和/或晕厥。在平均31个月的随访期间，所有患者起搏器植入后均未再出现症状。在平均21个月的随访中，未接受起搏器治疗的一度希氏束内延迟患者均未出现症状性缓慢性心律失常或晕厥。
