结核性脑膜炎导致抗利尿激素分泌过多。
Hypersecretion of anti-diuretic hormone due to tuberculous meningitis.
作者信息
Smith J, Godwin-Austen R
出版信息
Postgrad Med J. 1980 Jan;56(651):41-4. doi: 10.1136/pgmj.56.651.41.
Abstract
Three patients with tuberculous meningitis complicated by hypersecretion of anti-diuretic hormone are described. The recognition of this complication is important in the treatment and prognosis of tuberculous meningitis.
摘要
本文描述了3例结核性脑膜炎合并抗利尿激素分泌过多的患者。认识到这一并发症对于结核性脑膜炎的治疗和预后很重要。
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Br Med J. 1937 Jul 17;2(3993):111-3. doi: 10.1136/bmj.2.3993.111.
2
Salt losing conditions; the renal defect in tuberculous meningitis.失盐情况；结核性脑膜炎的肾脏缺陷。
J Lab Clin Med. 1951 Apr;37(4):550-61.
3
Total body chloride in tuberculous meningitis.结核性脑膜炎患者的全身氯化物含量
Pediatrics. 1954 Aug;14(2):87-92.
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Hyponatremia resulting from apparently inappropriate secretion of antidiuretic hormone in patients with pulmonary tuberculosis.肺结核患者因抗利尿激素分泌明显不当导致的低钠血症。
Am Rev Respir Dis. 1965 Oct;92(4):609-16. doi: 10.1164/arrd.1965.92.4.609.
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Antidiuretic principle in tuberculous lung tissue of a patient with pulmonary tuberculosis and hyponatremia.一名患有肺结核和低钠血症患者的结核性肺组织中的抗利尿物质
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