Case R B
Basic Res Cardiol. 1981 Jul-Aug;76(4):387-8. doi: 10.1007/BF01908328.
These data indicate that the observed PCO2 change following an abrupt change in myocardial O2 consumption, occurs too slowly to participate in the initial adjustment in metabolic coronary flow change, which is essentially complete within 30 sec. Also, the magnitude of PCO2 response is insufficient to conclude that CO2 can provide the major cause of flow regulation. However, CO2 could act as a back-up regulator. These conclusions may be modified if further information becomes available.
这些数据表明，心肌氧耗突然变化后观察到的PCO2变化发生得太慢，无法参与代谢性冠脉血流变化的初始调节，而这种调节在30秒内基本完成。此外，PCO2反应的幅度不足以得出二氧化碳是血流调节主要原因的结论。然而，二氧化碳可能作为一种备用调节器。如果有更多信息，这些结论可能会被修改。
