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Serum-sulfated lithocholate as an indicator of cholestasis during parenteral nutrition in infants and children.

作者信息

Farrell M K, Balistreri W F, Suchy F J

出版信息

JPEN J Parenter Enteral Nutr. 1982 Jan-Feb;6(1):30-3. doi: 10.1177/014860718200600130.

DOI:10.1177/014860718200600130
PMID:6804652
Abstract
摘要

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Serum-sulfated lithocholate as an indicator of cholestasis during parenteral nutrition in infants and children.血清硫酸化石胆酸作为婴幼儿肠外营养期间胆汁淤积的指标。
JPEN J Parenter Enteral Nutr. 1982 Jan-Feb;6(1):30-3. doi: 10.1177/014860718200600130.
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Laboratory monitoring of parenteral nutrition-associated hepatic dysfunction in infants.
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Reversal of parenteral nutrition-associated liver disease in two infants with short bowel syndrome using parenteral fish oil: implications for future management.使用肠外鱼油逆转两名短肠综合征婴儿的肠外营养相关肝病:对未来管理的启示
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Pathologic versus physiologic cholestasis: elevated serum concentration of a secondary bile acid in the presence of hepatobiliary disease.病理性胆汁淤积与生理性胆汁淤积:在肝胆疾病存在的情况下,继发性胆汁酸血清浓度升高。
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Hepatic failure in infants on total parenteral nutrition (TPN): clinical and histopathologic observations.
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Hepatic alterations during total parenteral nutrition in patients with inflammatory bowel disease: a possible consequence of lithocholate toxicity.炎症性肠病患者全胃肠外营养期间的肝脏改变:石胆酸毒性的一个可能后果。
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Withholding parenteral nutrition during critical illness increases plasma bilirubin but lowers the incidence of biliary sludge.在危重病期间停止肠外营养会增加血浆胆红素,但会降低胆泥的发生率。
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[Liver lesions and lipid metabolism. I. Lipid, bilirubin and enzyme activities in plasma in experimental cholestasis in rats].[肝脏病变与脂质代谢。I. 大鼠实验性胆汁淤积时血浆中的脂质、胆红素及酶活性]
Acta Hepatosplenol. 1970 Jan-Feb;17(1):36-47.

引用本文的文献

1
Cholesterol synthesis in the pathogenesis of lithocholic acid-induced cholestasis.胆固醇合成在石胆酸诱导的胆汁淤积发病机制中的作用
Lipids. 1988 Mar;23(3):230-3. doi: 10.1007/BF02535463.
2
Fetal and neonatal bile acid synthesis and metabolism--clinical implications.胎儿及新生儿胆汁酸的合成与代谢——临床意义
J Inherit Metab Dis. 1991;14(4):459-77. doi: 10.1007/BF01797918.