Ishikawa K, Satoh K
Arch Otorhinolaryngol. 1983;238(1):69-75. doi: 10.1007/BF00453743.
A bipolar stimulating electrode was chronically implanted at the mesodiencephalon in the rabbit to induce so-called central nystagmus. The nystagmus was remarkably facilitated in either darkness by blindfolding an eye ipsilateral to the stimulation site, but the nystagmus was almost never facilitated by blindfolding an eye contralateral to the stimulation site. This is a phenomenon related to photic inhibition. This phenomenon disappeared after ablation of the flocculus and paraflocculus ipsilateral to the stimulation site. The neural pathway responsible for it is considered to be by way of the flocculus.
在兔子的中脑间脑慢性植入一个双极刺激电极，以诱发所谓的中枢性眼球震颤。通过蒙住刺激部位同侧的眼睛，在黑暗中眼球震颤会显著增强，但蒙住刺激部位对侧的眼睛几乎不会使眼球震颤增强。这是一种与光抑制相关的现象。在切除刺激部位同侧的绒球和旁绒球后，这种现象消失。认为负责此现象的神经通路是通过绒球的。
