Pau H, Schmitt-Gräff A
Albrecht Von Graefes Arch Klin Exp Ophthalmol. 1980;214(4):220-38. doi: 10.1007/BF00417518.
Clinical signs and morphological evidence suggest a primary endothelial damage by the blood-clot in the anterior chamber, and, may be, by raised intraocular pressure, in blood-staining of the cornea. Corneal oedema develops, hemoglobin penetrate into the oedematous corneal stroma peripheral of the edge of Descemet's membrane (Abb. 1, 3). Multiple hemoglobin deposits without iron positive reaction appear in the corneal stroma (Abb. 2-5). Keratocytes take up these fragments and ferritine is formed in the lysosomes (Abb. 6).
临床体征和形态学证据表明，前房内的血凝块以及可能升高的眼压导致了角膜血染时的原发性内皮损伤。角膜水肿形成，血红蛋白渗入Descemet膜边缘周边的水肿角膜基质中（图1、3）。角膜基质中出现多个无铁阳性反应的血红蛋白沉积物（图2 - 5）。角膜细胞摄取这些碎片并在溶酶体中形成铁蛋白（图6）。
