Cristini G, Consolani A, Lazzari F
Albrecht Von Graefes Arch Klin Exp Ophthalmol. 1980;213(4):283-90. doi: 10.1007/BF00417551.
Some considerations of the vascular events occurring the uvea during the course of glaucoma are reported and a mathematical model is adopted to clarify the interdependence between intraocular pressure, rate of blood flow, blood pressure gradient, and the number of unobstructed capillary vessels in the uveal membrane. By means of this mathematical model the physiopathological vascular cause of the rise in intraocular pressure is confirmed (also independently of an obstacle to aqueous outflow), and the clinical implications in relation to visual sensory impairment are emphasized.
报告了青光眼病程中葡萄膜发生的血管事件的一些考量，并采用数学模型来阐明眼内压、血流速率、血压梯度以及葡萄膜中无阻塞毛细血管数量之间的相互依存关系。通过该数学模型，确认了眼内压升高的生理病理血管原因（也独立于房水流出障碍），并强调了与视觉感觉障碍相关的临床意义。
