Kühn W, Staemmler H J, Neckel E
Geburtshilfe Frauenheilkd. 1981 Oct;41(10):698-701. doi: 10.1055/s-2008-1037319.
The article deals with the various theories, hypotheses, localisations and experiments in connection with the genesis of uterine and extrauterine endometriosis. It is explained that there is not just one single valid cause; rather, it will be necessary to resort to various theories, depending on the localisation of the endometrial heterotopia. The article describes the case of a young woman with genital malformation combined with ovarial endometriosis. This malformation syndrome is known as Mayer-Rokitansky-Küster syndrome with congenital aplasia of the vagina and a rudimentary uterus bicornis solidus. Since such uteri do not contain any endometrium which is capable of functioning, this would exclude any canalicular, haematogenic, lymphogenic, embolic, metastatic or homotransplantational cause of endometriosis. In fact, ovarial endometrial tissue heterotopia is due to pluropotentiality of the coelomic epithelium and is produced by indentation of cortical epithelium and segmentation.
本文探讨了与子宫和子宫外子宫内膜异位症发生相关的各种理论、假说、定位及实验。文中指出,子宫内膜异位症并非由单一原因导致;相反,根据子宫内膜异位的位置,有必要参考多种理论。本文描述了一名患有生殖器畸形并伴有卵巢子宫内膜异位症的年轻女性病例。这种畸形综合征被称为梅耶-罗基坦斯基-库斯特综合征,其特征为先天性阴道发育不全和双角实性残角子宫。由于此类子宫不含任何有功能的子宫内膜,这就排除了子宫内膜异位症的任何管腔性、血源性、淋巴源性、栓塞性、转移性或同种移植性病因。实际上,卵巢子宫内膜组织异位是由于体腔上皮的多能性,由皮质上皮的凹陷和分割产生。