Göttinger W
Klin Monbl Augenheilkd. 1978;172(6):880-3.
In peripheral cystoid degenerations as the precursor of senile retinoschisis basement membrane material and collagen fibrils are formed; after retinal splitting this newly formed material together with ruptured tissue columns build up a layer on the inner surface of the outer wall of the retinoschisis which tends to shrinkage. This mechanism leads to hole formation in the outer wall; the holes consequently enlarge by a curling inward of the edge of the defect.
在作为老年性视网膜劈裂症前驱的周边囊样变性中，基底膜物质和胶原纤维形成；视网膜劈裂后，这种新形成的物质与破裂的组织柱在外壁内表面形成一层，该层易于收缩。这种机制导致在外壁形成孔洞；这些孔洞随后因缺损边缘向内卷曲而扩大。
