血管平滑肌细胞的前列腺素合成受缓激肽、哌唑嗪和肼苯哒嗪刺激。 - Suppr | 超能文献

Prostaglandin synthesis by vascular smooth muscle cells is stimulated by bradykinin, prazosin and hydralazine.

作者信息

Dyer R D, Huttner J J, Tan S Y, Mulrow P J

出版信息

Prog Lipid Res. 1981;20:557-60. doi: 10.1016/0163-7827(81)90099-0.

DOI:10.1016/0163-7827(81)90099-0

Abstract

摘要

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Prostaglandin synthesis by vascular smooth muscle cells is stimulated by bradykinin, prazosin and hydralazine.血管平滑肌细胞的前列腺素合成受缓激肽、哌唑嗪和肼苯哒嗪刺激。

Prog Lipid Res. 1981;20:557-60. doi: 10.1016/0163-7827(81)90099-0.

2

Fatty acid metabolism and cell proliferation. III. Effect of prostaglandin biosynthesis either from exogenous fatty acid or endogenous fatty acid release with hydralazine.脂肪酸代谢与细胞增殖。III. 外源性脂肪酸或肼屈嗪释放的内源性脂肪酸生成前列腺素的作用。

Lipids. 1983 May;18(5):349-52. doi: 10.1007/BF02537230.

3

Action of bradykinin at the cyclooxygenase step in prostanoid synthesis through the arachidonic acid cascade.缓激肽在通过花生四烯酸级联反应进行的前列腺素合成中，对环氧化酶步骤的作用。

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Mechanism of bradykinin-stimulated prostacyclin synthesis in porcine aortic endothelial cells.缓激肽刺激猪主动脉内皮细胞合成前列环素的机制。

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5

Hydralazine inhibits vascular reactivity by a mechanism independent of vascular prostaglandin biosynthesis: role of thromboxane synthetase in blocking hydralazine actions.肼屈嗪通过一种独立于血管前列腺素生物合成的机制抑制血管反应性：血栓素合成酶在阻断肼屈嗪作用中的作用。

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Bradykinin-stimulated prostaglandin E2 production by endothelial cells and its modulation by antiinflammatory compounds.缓激肽刺激内皮细胞产生前列腺素E2及其受抗炎化合物的调节作用。

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Expression of 5-HT2- and bradykinin B2-receptors coupled to phosphoinositide hydrolysis in cultured guinea-pig aortic smooth muscle cells.豚鼠主动脉平滑肌细胞中与磷酸肌醇水解偶联的5-羟色胺2型和缓激肽B2型受体的表达

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Comparison of haemodynamic effects of oral prazosin, oral hydralazine, and intravenous nitroprusside in same patients with chronic heart failure.口服哌唑嗪、口服肼屈嗪和静脉注射硝普钠对同一组慢性心力衰竭患者血流动力学影响的比较。

Br Heart J. 1979 Dec;42(6):664-70. doi: 10.1136/hrt.42.6.664.

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Comparison of haemodynamic effects of oral hydralazine and prazosin hydrochloride in patients with chronic congestive heart failure.口服肼屈嗪和盐酸哌唑嗪对慢性充血性心力衰竭患者血流动力学影响的比较。

Br Heart J. 1979 Dec;42(6):657-63. doi: 10.1136/hrt.42.6.657.

引用本文的文献

1

Fatty acid metabolism and cell proliferation. III. Effect of prostaglandin biosynthesis either from exogenous fatty acid or endogenous fatty acid release with hydralazine.脂肪酸代谢与细胞增殖。III. 外源性脂肪酸或肼屈嗪释放的内源性脂肪酸生成前列腺素的作用。

Lipids. 1983 May;18(5):349-52. doi: 10.1007/BF02537230.

2

Effect of des arginine9-bradykinin and other bradykinin fragments on the synthesis of prostacyclin and the binding of bradykinin by vascular cells in culture.去精氨酸9-缓激肽及其他缓激肽片段对培养的血管细胞中前列环素合成及缓激肽结合的影响。

Agents Actions. 1988 Jul;24(3-4):224-31. doi: 10.1007/BF02028275.

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Clinical pharmacokinetics and therapeutic use of hydralazine in congestive heart failure.肼屈嗪在充血性心力衰竭中的临床药代动力学及治疗应用

Clin Pharmacokinet. 1989 Feb;16(2):86-9. doi: 10.2165/00003088-198916020-00003.