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Origin and function of suppressor macrophages in myeloma.

作者信息

Kennard J, Zolla-Pazner S

出版信息

J Immunol. 1980 Jan;124(1):268-73.

PMID:6965292
Abstract
摘要

相似文献

1
Origin and function of suppressor macrophages in myeloma.骨髓瘤中抑制性巨噬细胞的起源与功能
J Immunol. 1980 Jan;124(1):268-73.
2
Regulation of myeloma growth in vitro by idiotype-specific T lymphocytes.独特型特异性T淋巴细胞对骨髓瘤体外生长的调节
J Immunol. 1980 Jan;124(1):424-30.
3
Antigen-specific regulation of myeloma cell differentiation in vivo by carrier-specific T cell factors and macrophages.载体特异性T细胞因子和巨噬细胞对体内骨髓瘤细胞分化的抗原特异性调节。
J Immunol. 1978 Sep;121(3):1066-74.
4
Immunosuppression in BALB/c mice bearing the plasmacytoma TEPC-183: massive increase in red pulp macrophages induced by the tumour.携带浆细胞瘤TEPC - 183的BALB/c小鼠的免疫抑制:肿瘤诱导红髓巨噬细胞大量增加。
Immunology. 1980 Jun;40(2):223-8.
5
Mechanism of neonatal idiotype suppression. III. Delayed maturation of plasmacytoma stem cells in neonatally suppressed hosts.新生儿独特型抑制机制。III. 新生期受抑制宿主中浆细胞瘤干细胞的延迟成熟
J Immunol. 1981 Aug;127(2):509-13.
6
Immunosuppression in plasmacytoma: induction of suppressor cells.浆细胞瘤中的免疫抑制:抑制细胞的诱导。
Clin Exp Immunol. 1982 Jan;47(1):191-6.
7
Lymphocyte surface membrane immunoglobulin in myeloma. I. M315-bearing T lymphocytes in mice with MOPC-315.骨髓瘤中的淋巴细胞表面膜免疫球蛋白。I. 携带MOPC - 315的小鼠中带有M315的T淋巴细胞
J Immunol. 1979 Sep;123(3):1110-6.
8
Immunoregulation of murine myeloma cell growth and differentiation: a monoclonal model of B cell differentiation.小鼠骨髓瘤细胞生长与分化的免疫调节:B细胞分化的单克隆模型
Immunol Rev. 1979;48:45-80. doi: 10.1111/j.1600-065x.1979.tb00298.x.
9
Suppressor cell activity in tumor-bearing mice. II. Inhibition of DNA synthesis and DNA polymerases by TBH splenic suppressor cells.荷瘤小鼠中的抑制细胞活性。II. 荷瘤小鼠脾脏抑制细胞对DNA合成和DNA聚合酶的抑制作用
J Immunol. 1978 Apr;120(4):1354-61.
10
Cyclical changes in susceptibility of a myeloma tumor (LPC-1) to immune destruction. II. Periodic fluctuations during growth in normal and nude mice and in culture.骨髓瘤肿瘤(LPC-1)对免疫破坏敏感性的周期性变化。II. 在正常小鼠、裸鼠体内生长及体外培养过程中的周期性波动
J Immunol. 1979 Mar;122(3):959-64.

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1
Low intensity vibration mitigates tumor progression and protects bone quantity and quality in a murine model of myeloma.低强度振动可减轻骨髓瘤小鼠模型中的肿瘤进展,并保护骨量和骨质。
Bone. 2016 Sep;90:69-79. doi: 10.1016/j.bone.2016.05.014. Epub 2016 Jun 2.
2
The relationship between hypogammaglobulinemia, monoclonal gammopathy of undetermined significance and humoral immunodeficiency: a case series.低丙种球蛋白血症、意义未明的单克隆丙种球蛋白血症和体液免疫缺陷之间的关系:病例系列。
J Clin Immunol. 2011 Oct;31(5):737-43. doi: 10.1007/s10875-011-9548-0. Epub 2011 Jun 5.
3
Mechanisms of polyclonal hypogammaglobulinaemia in multiple myeloma (MM).
多发性骨髓瘤(MM)中多克隆低丙种球蛋白血症的机制。
Med Oncol. 1999 Jul;16(2):73-7. doi: 10.1007/BF02785839.
4
Alternatively activated macrophages actively inhibit proliferation of peripheral blood lymphocytes and CD4+ T cells in vitro.替代性活化的巨噬细胞在体外可有效抑制外周血淋巴细胞和CD4 + T细胞的增殖。
Immunology. 1997 Dec;92(4):478-86. doi: 10.1046/j.1365-2567.1997.00371.x.
5
Immunotherapy of a plasmacytoma with attenuated salmonella.用减毒沙门氏菌对浆细胞瘤进行免疫治疗。
Med Oncol. 1995 Jun;12(2):103-8. doi: 10.1007/BF01676710.
6
PWM-induced generation of immunoglobulin-secreting cells in patients with multiple myeloma.多发性骨髓瘤患者中 PWM 诱导的免疫球蛋白分泌细胞的产生。
Klin Wochenschr. 1981 Oct 1;59(19):1101-8. doi: 10.1007/BF01746197.
7
Characterization of the cytotoxin produced by macrophages in response to dengue virus-induced cytotoxic factor.巨噬细胞响应登革病毒诱导的细胞毒性因子产生的细胞毒素的特性分析。
Br J Exp Pathol. 1983 Apr;64(2):185-90.
8
Soluble suppressor factors in patients with acquired immune deficiency syndrome and its prodrome. Elaboration in vitro by T lymphocyte-adherent cell interactions.获得性免疫缺陷综合征患者及其前驱期患者的可溶性抑制因子。通过T淋巴细胞与黏附细胞相互作用在体外的阐述。
J Clin Invest. 1983 Dec;72(6):2072-81. doi: 10.1172/JCI111172.
9
T-cell imbalances in patients with multiple myeloma: an analysis by monoclonal antibodies.多发性骨髓瘤患者的T细胞失衡:单克隆抗体分析
J Clin Immunol. 1983 Jul;3(3):277-84. doi: 10.1007/BF00915352.
10
Suppression of antitumor immunity by macrophages in spleens of mice bearing a large MOPC-315 tumor.携带大型MOPC-315肿瘤的小鼠脾脏中巨噬细胞对抗肿瘤免疫的抑制作用。
Cancer Immunol Immunother. 1984;16(3):162-9. doi: 10.1007/BF00205423.