Hendricks I M, Holliday I E, Ruddock K H
Brain. 1981 Dec;104(Pt 4):813-40. doi: 10.1093/brain/104.4.813.
This paper deals with a single male subject, M., who has a well-defined visual defect of a kind not previously described. The defect is associated with a potent inhibitory response to long-wavelength stimuli, which spreads for up to 12 deg from the area of stimulation, suppressing M.'s detection of other high contrast stimuli. The effect can also be observed, weakly, with certain other coloured stimuli, but not with yellows or whites. Detailed psychophysical data are given for threshold sensitivity, colour matching, two-colour increment thresholds and visual acuity, all of which are more or less abnormal, depending on the stimulus colour. No abnormal effects were, however, observed with white light stimuli. On the basis of M.'s ability to fuse red and green random dot stereogram pairs, we argue that the inhibition arises centrally in the visual pathways. and we examine the implications of M.'s response characteristics for the analysis of central visual processing.
本文研究的是一位男性受试者M，他患有一种此前未被描述过的明确视觉缺陷。该缺陷与对长波长刺激的强烈抑制反应有关，这种抑制反应从刺激区域扩散至多达12度，抑制了M对其他高对比度刺激的检测。在某些其他颜色的刺激下也能微弱地观察到这种效应，但黄色或白色刺激则不会。文中给出了关于阈值敏感性、颜色匹配、双色增量阈值和视力的详细心理物理学数据，所有这些数据或多或少都存在异常，具体取决于刺激颜色。然而，在白光刺激下未观察到异常效应。基于M融合红绿随机点立体图对的能力，我们认为这种抑制作用产生于视觉通路的中枢，并且我们探讨了M的反应特征对中枢视觉处理分析的影响。
